Statements in which the resource exists.
SubjectPredicateObjectContext
http://linkedlifedata.com/r...rdf:typeentrezgene:Interactionlld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:15972662lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:10430052lld:entrezgene
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http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:11287575lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:15113900lld:entrezgene
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http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:16188969lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:8976200lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:12787886lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:20835359lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:12482395lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:10820198lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:9653130lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:10872839lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:20817073lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:9826729lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:16343773lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:10486323lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:11162626lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:12834106lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:15710459lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:17507482lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:20181695lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:20929726lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:20967243lld:entrezgene
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http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:22101518lld:entrezgene
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http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:9744279lld:entrezgene
http://linkedlifedata.com/r...entrezgene:pubmedpubmed-article:22954308lld:entrezgene
http://linkedlifedata.com/r...entrezgene:interactantentrez-gene:155971lld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextThe fusion of insulin-like growth factor I (IGF I) with stromal cell-derived factor I or alpha1 proteinase inhibitor has the capacity to compete with the binding of HIV-1 gp120 to CD4 receptorlld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextSDF-1alpha reverses gp120-induced downregulation of CD79b in CD40- and IL-4-activated purified HIV-1 seronegative human peripheral blood B cellslld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextHIV-1 gp120 from a T-cell-tropic virus causes CD4-dependent antagonism of CXCR4 response to SDF-1alpha, whereas gp120 from macrophage-tropic viruses causes CD4-dependent antagonism of CCR5 response to MIP-1alphalld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextA synthetic peptide domain of the V3 region of HIV-1 gp120 activates the FPRL1 receptor in monocytes and neutrophils and causes reduced response to several chemokines that use multiple cell receptors, including SDF-1alpha and RANTESlld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextChemokines such as fractalkine, macrophage-derived chemokine (MDC), RANTES, and SDF-1alpha are able to block gp120-induced apoptosis of hippocampal neurons; both fractalkine and MDC activate ERK-1/2, while SDF-1alpha activates CREBlld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextRANTES, stromal derived factor-1alpha (SDF-1alpha), macrophage-derived chemokine (MDC), and their combination attenuate HIV-1 gp120-induced food and water intake decrease in ratslld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextCXCR4-tropic and CXCR4/CCR5 dual-tropic HIV-1 gp120 proteins inhibit B cell chemotaxis toward CXCL12, CCL20, and CCL21, and gp120-induced p38 MAPK activation is triggered by CCL21 and CCL20lld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextThe entry of T cell-tropic HIV-1 isolates into cells is blocked by SDF-1, which interacts with the HIV-1 gp120 coreceptor CXCR4lld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextApoptosis of CD8+ T cells is mediated by the interaction between TNF-alpha bound to the membrane of macrophages (mbTNF) and a receptor on CD8+ T cells (TNF-receptor II, or TNFRII), which is upregulated by treatment with recombinant HIV-1 gp120 or SDF-1lld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextThe death rate of CD8+ T cells by apoptosis, which is induced by HIV-1 gp120 from CXCR4-tropic HIV strains or by the ligand of the chemokine receptor CXCR4 (SDF-1), increased markedly during HIV infection of peripheral blood mononuclear cellslld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextCXCL12 variant proteins exhibits the various levels of inhibition of HIV-1 X4 Env-mediated fusion. The strongest and weakest inhibition activities among the variant proteins in the X4 Env fusion assay are CXCL12gamma and CXCL12delta, respectivelylld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextThe ability of HIV-1 gp120 to inhibit SDF-1a-induced chemotaxis is mediated by the CD4 receptor and Lck signalinglld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextCXCL12 and HIV-1 gp120 modulate the excitability of Cajal-Retzius cells in opposite directionslld:entrezgene
http://linkedlifedata.com/r...entrezgene:geneRifTextHIV-1 gp120 abnormally interferes with SDF-1-mediated T cell chemotaxis and cell migration in resting CD4+ T Cellslld:entrezgene
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