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pubmed-article:9790822pubmed:abstractTextSmall intestine microvascular vasoconstriction and hypoperfusion develop after resuscitation (RES) from hemorrhage (HEM), despite restoration of central hemodynamics. The responsible mechanisms are unclear. We hypothesized that the microvascular impairment following HEM/RES was due to decreased intestinal microvascular nitric oxide (NO) production.lld:pubmed
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pubmed-article:9790822pubmed:articleTitleSmall intestinal production of nitric oxide is decreased following resuscitated hemorrhage.lld:pubmed
pubmed-article:9790822pubmed:affiliationDepartment of Surgery, University of Louisville School of Medicine, Louisville, Kentucky, 40292, USA.lld:pubmed
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