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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
1998-12-4
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pubmed:abstractText |
Small intestine microvascular vasoconstriction and hypoperfusion develop after resuscitation (RES) from hemorrhage (HEM), despite restoration of central hemodynamics. The responsible mechanisms are unclear. We hypothesized that the microvascular impairment following HEM/RES was due to decreased intestinal microvascular nitric oxide (NO) production.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
0022-4804
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pubmed:author |
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pubmed:copyrightInfo |
Copyright 1998 Academic Press.
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pubmed:issnType |
Print
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pubmed:volume |
80
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
102-9
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:9790822-Animals,
pubmed-meshheading:9790822-Gastrointestinal Hemorrhage,
pubmed-meshheading:9790822-Hemodynamics,
pubmed-meshheading:9790822-Immunohistochemistry,
pubmed-meshheading:9790822-Intestine, Small,
pubmed-meshheading:9790822-Male,
pubmed-meshheading:9790822-Nitric Oxide,
pubmed-meshheading:9790822-Rats,
pubmed-meshheading:9790822-Rats, Sprague-Dawley,
pubmed-meshheading:9790822-Resuscitation,
pubmed-meshheading:9790822-von Willebrand Factor
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pubmed:year |
1998
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pubmed:articleTitle |
Small intestinal production of nitric oxide is decreased following resuscitated hemorrhage.
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pubmed:affiliation |
Department of Surgery, University of Louisville School of Medicine, Louisville, Kentucky, 40292, USA.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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