| pubmed-article:9724773 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9724773 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:9724773 | lifeskim:mentions | umls-concept:C0003467 | lld:lifeskim |
| pubmed-article:9724773 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
| pubmed-article:9724773 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:9724773 | pubmed:issue | 18 | lld:pubmed |
| pubmed-article:9724773 | pubmed:dateCreated | 1998-9-28 | lld:pubmed |
| pubmed-article:9724773 | pubmed:abstractText | Brain serotonin (5-HT) has been implicated in a number of physiological processes and pathological conditions. These effects are mediated by at least 14 different 5-HT receptors. We have inactivated the gene encoding the 5-HT1A receptor in mice and found that receptor-deficient animals have an increased tendency to avoid a novel and fearful environment and to escape a stressful situation, behaviors consistent with an increased anxiety and stress response. Based on the role of the 5-HT1A receptor in the feedback regulation of the 5-HT system, we hypothesize that an increased serotonergic neurotransmission is responsible for the anxiety-like behavior of receptor-deficient animals. This view is consistent with earlier studies showing that pharmacological activation of the 5-HT system is anxiogenic in animal models and also in humans. | lld:pubmed |
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| pubmed-article:9724773 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:9724773 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9724773 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9724773 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:9724773 | pubmed:issn | 0027-8424 | lld:pubmed |
| pubmed-article:9724773 | pubmed:author | pubmed-author:ShenkTT | lld:pubmed |
| pubmed-article:9724773 | pubmed:author | pubmed-author:TothMM | lld:pubmed |
| pubmed-article:9724773 | pubmed:author | pubmed-author:RobinsonP SPS | lld:pubmed |
| pubmed-article:9724773 | pubmed:author | pubmed-author:ParksC LCL | lld:pubmed |
| pubmed-article:9724773 | pubmed:author | pubmed-author:SibilleEE | lld:pubmed |
| pubmed-article:9724773 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9724773 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:9724773 | pubmed:volume | 95 | lld:pubmed |
| pubmed-article:9724773 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9724773 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9724773 | pubmed:pagination | 10734-9 | lld:pubmed |
| pubmed-article:9724773 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:9724773 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9724773 | pubmed:articleTitle | Increased anxiety of mice lacking the serotonin1A receptor. | lld:pubmed |
| pubmed-article:9724773 | pubmed:affiliation | Howard Hughes Medical Institute, Department of Molecular Biology, Princeton University, Princeton, NJ 08544-1014, USA. | lld:pubmed |
| pubmed-article:9724773 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9724773 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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