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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
7-8
|
pubmed:dateCreated |
1998-10-9
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pubmed:abstractText |
1. Endothelial dysfunction, due to reductions in nitric oxide (NO) action, is an early feature of macrovascular disease. 2. Non-invasive measurement of endothelial function may be assessed by postischaemic dilation of forearm vessels, using plethysmography, or flow-mediated dilatation of the brachial artery, using ultrasound. 3. Brachial flow-mediated dilatation reflects NO release and/or action more than forearm hyperaemia. 4. These techniques have been used as surrogate measures of coronary endothelial function. 5. Methodological, physiological and clinical aspects of the techniques are discussed.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
|
pubmed:chemical | |
pubmed:status |
MEDLINE
|
pubmed:issn |
0305-1870
|
pubmed:author | |
pubmed:issnType |
Print
|
pubmed:volume |
25
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
640-3
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading | |
pubmed:articleTitle |
Special article: non-invasive measurement of endothelial function.
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pubmed:affiliation |
University Department of Medicine, Royal Perth Hospital, Western Australia, Australia. daviplay@cyllene.uwa.edu.au
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pubmed:publicationType |
Journal Article,
Review,
Research Support, Non-U.S. Gov't
|