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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
|
pubmed:dateCreated |
1998-5-14
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pubmed:abstractText |
Retroviruses have repeatedly been suggested as a possible trigger mechanism for systemic lupus erythematosus (SLE). We review the role of human immunodeficiency virus (HIV)-1 envelope glycoprotein (gp120) in the induction of immune dysregulation including autoimmunity in HIV-1 infection, and discuss the possible relationship between HIV and SLE in the retroviral etiology.
|
pubmed:language |
eng
|
pubmed:journal | |
pubmed:citationSubset |
IM
|
pubmed:chemical | |
pubmed:status |
MEDLINE
|
pubmed:issn |
0392-856X
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pubmed:author | |
pubmed:issnType |
Print
|
pubmed:volume |
16
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
175-80
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pubmed:dateRevised |
2005-11-16
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pubmed:meshHeading |
pubmed-meshheading:9536396-Acquired Immunodeficiency Syndrome,
pubmed-meshheading:9536396-HIV Envelope Protein gp120,
pubmed-meshheading:9536396-Humans,
pubmed-meshheading:9536396-Immune System,
pubmed-meshheading:9536396-Lupus Erythematosus, Systemic,
pubmed-meshheading:9536396-Retroviridae Infections,
pubmed-meshheading:9536396-Tumor Necrosis Factor-alpha
|
pubmed:articleTitle |
HIV infection and SLE: their pathogenic relationship.
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pubmed:affiliation |
Department of Medicine, Juntendo University, Izu-Nagaoka Hospital, Shizuoka, Japan.
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pubmed:publicationType |
Journal Article,
Review
|