Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
1998-4-13
pubmed:abstractText
A recent view of atherosclerosis indicates vascular smooth muscle cell proliferation not only as a response-to-injury but also as an essential reparative process leading to plaque stabilization. To assess the role of vascular smooth muscle cell in vasculitides we studied, as a model, 10 cases of Horton's arteritis. In particular, we investigated by immunohistochemical analysis cellular population and proliferation rate in the intimal layer. As in atherosclerotic plaques, intimal thickening in arteritis is essentially due to proliferation and migration of vascular smooth muscle cells. We suppose that, whatever the vascular disease, intimal hyperplasia contributes to stability of the vessel's wall; however the reactive/reparative process is extremely slow in spontaneous atherosclerosis, accelerated in arteritis.
pubmed:language
ita
pubmed:journal
pubmed:citationSubset
IM
pubmed:status
MEDLINE
pubmed:month
Oct
pubmed:issn
0031-2983
pubmed:author
pubmed:issnType
Print
pubmed:volume
89
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
491-4
pubmed:dateRevised
2007-11-15
pubmed:meshHeading
pubmed:year
1997
pubmed:articleTitle
[The reactive and/or repair role of the smooth muscle cells in Horton's arteritis].
pubmed:affiliation
Istituto di Anatomia e Istologia Patologica, Università di Firenze.
pubmed:publicationType
Journal Article, Comparative Study, English Abstract