| pubmed-article:9434765 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9434765 | lifeskim:mentions | umls-concept:C0205245 | lld:lifeskim |
| pubmed-article:9434765 | lifeskim:mentions | umls-concept:C1415629 | lld:lifeskim |
| pubmed-article:9434765 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
| pubmed-article:9434765 | lifeskim:mentions | umls-concept:C0301039 | lld:lifeskim |
| pubmed-article:9434765 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:9434765 | pubmed:dateCreated | 1998-2-3 | lld:pubmed |
| pubmed-article:9434765 | pubmed:abstractText | CBP functions as a key transcriptional coactivator for a variety of transcription factors. We show here that the hepatocyte nuclear factor 4 (HNF4), a transcription factor in the nuclear receptor superfamily with no defined ligand, is cloned by yeast two-hybrid system using CBP as a bait. GST-pull down assay with nuclear extracts or in vitro translation products revealed that CBP and HNF4 interact with each other at the middle portion (aa 119-375) of HNF4 and two distinct regions (aa 271-451 and 1626-2259) of CBP, respectively, in the ligand-independent manner. Co-transfection experiments indicated that CBP is capable of activating HNF4 site-mediated transcription. These results suggested a functional association between CBP and HNF4 in trans-activation. | lld:pubmed |
| pubmed-article:9434765 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9434765 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9434765 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9434765 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9434765 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:9434765 | pubmed:issn | 0006-291X | lld:pubmed |
| pubmed-article:9434765 | pubmed:author | pubmed-author:MurakamiKK | lld:pubmed |
| pubmed-article:9434765 | pubmed:author | pubmed-author:FukamizuAA | lld:pubmed |
| pubmed-article:9434765 | pubmed:author | pubmed-author:YoshidaEE | lld:pubmed |
| pubmed-article:9434765 | pubmed:author | pubmed-author:TakiguchiMM | lld:pubmed |
| pubmed-article:9434765 | pubmed:author | pubmed-author:ArataniSS | lld:pubmed |
| pubmed-article:9434765 | pubmed:author | pubmed-author:MiyagishiMM | lld:pubmed |
| pubmed-article:9434765 | pubmed:author | pubmed-author:ItouHH | lld:pubmed |
| pubmed-article:9434765 | pubmed:author | pubmed-author:OsumuTT | lld:pubmed |
| pubmed-article:9434765 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9434765 | pubmed:day | 29 | lld:pubmed |
| pubmed-article:9434765 | pubmed:volume | 241 | lld:pubmed |
| pubmed-article:9434765 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9434765 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9434765 | pubmed:pagination | 664-9 | lld:pubmed |
| pubmed-article:9434765 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:9434765 | pubmed:year | 1997 | lld:pubmed |
| pubmed-article:9434765 | pubmed:articleTitle | Functional association between CBP and HNF4 in trans-activation. | lld:pubmed |
| pubmed-article:9434765 | pubmed:affiliation | Institute of Applied Biochemistry, University of Tsukuba, Ibaraki, Japan. | lld:pubmed |
| pubmed-article:9434765 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9434765 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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