Linked Life Data

8896062

Source:http://linkedlifedata.com/resource/pubmed/id/8896062

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
7-8
pubmed:dateCreated
1997-2-13
pubmed:abstractText
Irrespective of the underlying mechanisms, joint pain usually originates in activation of nociceptors, or free nerve endings. Nociceptive signals release a large number of neuromediators, such as substance P and the calcitonin gene-related peptide. Complex neuronal activation occurs, which involves not only local sensitization of joint nociceptors but also modifications in central pain pathways. Additional complexity results from the ability of numerous environmental, psychological, and constitutional factors to influence joint pain.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:status
MEDLINE
pubmed:issn
1169-8446
pubmed:author
pubmed:issnType
Print
pubmed:volume
63
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
485-92
pubmed:dateRevised
2005-11-16
pubmed:meshHeading
pubmed:articleTitle
Pathophysiology of joint pain.
pubmed:affiliation
Rheumatology Department A and Pain Clinic, Cochin Teaching Hospital, Paris, France.
pubmed:publicationType
Journal Article, Review