pubmed-article:8733232 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:8733232 | lifeskim:mentions | umls-concept:C0038435 | lld:lifeskim |
pubmed-article:8733232 | lifeskim:mentions | umls-concept:C0004595 | lld:lifeskim |
pubmed-article:8733232 | lifeskim:mentions | umls-concept:C0040648 | lld:lifeskim |
pubmed-article:8733232 | lifeskim:mentions | umls-concept:C0029073 | lld:lifeskim |
pubmed-article:8733232 | lifeskim:mentions | umls-concept:C0205246 | lld:lifeskim |
pubmed-article:8733232 | lifeskim:mentions | umls-concept:C2613267 | lld:lifeskim |
pubmed-article:8733232 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
pubmed-article:8733232 | lifeskim:mentions | umls-concept:C1554184 | lld:lifeskim |
pubmed-article:8733232 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:8733232 | pubmed:dateCreated | 1996-12-10 | lld:pubmed |
pubmed-article:8733232 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8733232 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8733232 | pubmed:abstractText | The sigma B transcription factor of Bacillus subtilis is activated in response to a variety of environmental stresses, including those imposed by entry into the stationary-growth phase, and by heat, salt or ethanol challenge to logarithmically growing cells. Although sigma B is thought to control a general stress regulon, the range of cellular functions it directs remains largely unknown. Our approach to understand the physiological role of sigma B is to characterize genes that require this factor for all or part of their expression, i.e. the csb genes. In this study, we report that the transposon insertion csb40::Tn917lac identifies an operon with three open reading frames, the second of which resembles plant proteins induced by desiccation stress. Primer-extension and operon-fusion experiments showed that the csb40 operon has a sigma B-dependent promoter which is strongly induced by the addition of salt to logarithmically growing cells. The csb40 operon also has a second, sigma H-dependent promoter that is unaffected by salt addition. These results provide support for the hypothesis that sigma B controls a general stress regulon, and indicate that the sigma B and sigma H regulons partly overlap. We suggest that in addition to its acknowledged role in the sporulation process, sigma H is also involved in controlling a subclass of genes that are broadly involved in a general stress response. | lld:pubmed |
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pubmed-article:8733232 | pubmed:language | eng | lld:pubmed |
pubmed-article:8733232 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8733232 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:8733232 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:8733232 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:8733232 | pubmed:month | Apr | lld:pubmed |
pubmed-article:8733232 | pubmed:issn | 0950-382X | lld:pubmed |
pubmed-article:8733232 | pubmed:author | pubmed-author:PriceC WCW | lld:pubmed |
pubmed-article:8733232 | pubmed:author | pubmed-author:BrodyM SMS | lld:pubmed |
pubmed-article:8733232 | pubmed:author | pubmed-author:VarónDD | lld:pubmed |
pubmed-article:8733232 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:8733232 | pubmed:volume | 20 | lld:pubmed |
pubmed-article:8733232 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:8733232 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:8733232 | pubmed:pagination | 339-50 | lld:pubmed |
pubmed-article:8733232 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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pubmed-article:8733232 | pubmed:meshHeading | pubmed-meshheading:8733232-... | lld:pubmed |
pubmed-article:8733232 | pubmed:year | 1996 | lld:pubmed |
pubmed-article:8733232 | pubmed:articleTitle | Bacillus subtilis operon under the dual control of the general stress transcription factor sigma B and the sporulation transcription factor sigma H. | lld:pubmed |
pubmed-article:8733232 | pubmed:affiliation | Department of Food Science and Technology, University of California, Davis 95616, USA. | lld:pubmed |
pubmed-article:8733232 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:8733232 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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