rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
5
|
pubmed:dateCreated |
1996-6-5
|
pubmed:abstractText |
Helicobacter pylori infection and duodenal ulcer disease are firmly correlated. However, the bacteria do mainly colonize the antrum, indicating an indirect pathogenic mechanism. The aim of this study was to test a concept claiming that H. pylori infection of the antrum selectively blocks normal inhibitory reflex pathways to gastrin and parietal cells.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
May
|
pubmed:issn |
0016-5085
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
110
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1386-94
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:8613042-Adult,
pubmed-meshheading:8613042-Aged,
pubmed-meshheading:8613042-Duodenal Ulcer,
pubmed-meshheading:8613042-Female,
pubmed-meshheading:8613042-Gastric Acid,
pubmed-meshheading:8613042-Gastric Dilatation,
pubmed-meshheading:8613042-Gastrin-Releasing Peptide,
pubmed-meshheading:8613042-Gastrins,
pubmed-meshheading:8613042-Helicobacter Infections,
pubmed-meshheading:8613042-Helicobacter pylori,
pubmed-meshheading:8613042-Humans,
pubmed-meshheading:8613042-Male,
pubmed-meshheading:8613042-Middle Aged,
pubmed-meshheading:8613042-Parietal Cells, Gastric,
pubmed-meshheading:8613042-Pentagastrin,
pubmed-meshheading:8613042-Peptides,
pubmed-meshheading:8613042-Pyloric Antrum,
pubmed-meshheading:8613042-Stomach Diseases
|
pubmed:year |
1996
|
pubmed:articleTitle |
A mechanism by which Helicobacter pylori infection of the antrum contributes to the development of duodenal ulcer.
|
pubmed:affiliation |
Department of Surgery, Sahlgrenska Hospital, Göteborg, Sweden.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|