| pubmed-article:8557193 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8557193 | lifeskim:mentions | umls-concept:C0008546 | lld:lifeskim |
| pubmed-article:8557193 | lifeskim:mentions | umls-concept:C0162493 | lld:lifeskim |
| pubmed-article:8557193 | lifeskim:mentions | umls-concept:C1533691 | lld:lifeskim |
| pubmed-article:8557193 | lifeskim:mentions | umls-concept:C0917720 | lld:lifeskim |
| pubmed-article:8557193 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:8557193 | pubmed:dateCreated | 1996-2-23 | lld:pubmed |
| pubmed-article:8557193 | pubmed:abstractText | NF-kappa B is a potent inducible transcription factor that regulates many genes in activated T cells. In this report we examined the ability of different subunits of NF-kappa B to enhance HIV-1 transcription in vitro with chromatin templates. We find that the p65 subunit of NF-kappa B is a strong transcriptional activator of nucleosome-assembled HIV-1 DNA, whereas p50 does not activate transcription, and that p65 activates transcription synergistically with Sp1 and distal HIV-1 enhancer-binding factors (LEF-1, Ets-1, and TFE-3). These effects were observed with chromatin, but not with nonchromatin templates. Furthermore, binding of either p50 or p65 with Sp1 induces rearrangement of the chromatin to a structure that resembles the one reported previously for integrated HIV-1 proviral DNA in vivo. These results suggest that p50 and Sp1 contribute to the establishment of the nucleosomal arrangement of the uninduced provirus in resting T cells, and that p65 activates transcription by recruitment of the RNA polymerase II transcriptional machinery to the chromatin-repressed basal promoter. | lld:pubmed |
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| pubmed-article:8557193 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:8557193 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8557193 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8557193 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:8557193 | pubmed:issn | 0890-9369 | lld:pubmed |
| pubmed-article:8557193 | pubmed:author | pubmed-author:LINNL BLB | lld:pubmed |
| pubmed-article:8557193 | pubmed:author | pubmed-author:JonesK AKA | lld:pubmed |
| pubmed-article:8557193 | pubmed:author | pubmed-author:CaoZZ | lld:pubmed |
| pubmed-article:8557193 | pubmed:author | pubmed-author:KadonagaJ TJT | lld:pubmed |
| pubmed-article:8557193 | pubmed:author | pubmed-author:PazinM JMJ | lld:pubmed |
| pubmed-article:8557193 | pubmed:author | pubmed-author:SheridanP LPL | lld:pubmed |
| pubmed-article:8557193 | pubmed:author | pubmed-author:CannonKK | lld:pubmed |
| pubmed-article:8557193 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8557193 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:8557193 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:8557193 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8557193 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8557193 | pubmed:pagination | 37-49 | lld:pubmed |
| pubmed-article:8557193 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:8557193 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8557193 | pubmed:articleTitle | NF-kappa B-mediated chromatin reconfiguration and transcriptional activation of the HIV-1 enhancer in vitro. | lld:pubmed |
| pubmed-article:8557193 | pubmed:affiliation | Department of Biology, University of California, San Diego (UCSD), La Jolla 92093-0347, USA. | lld:pubmed |
| pubmed-article:8557193 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8557193 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:8557193 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:8557193 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:8557193 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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