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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
5
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pubmed:dateCreated |
1993-6-11
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pubmed:abstractText |
Cardiac cells display rate-dependent beat-to-beat variations in action-potential duration (APD), action potential amplitude (APA), and excitability during periodic stimulation. We hypothesized that quinidine causes a marked increase in the variability of APD, APA, and excitability of ventricular cells isolated from quinidine-toxic, arrhythmic ventricles.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
0009-7322
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
87
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1661-72
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pubmed:dateRevised |
2010-3-24
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pubmed:meshHeading |
pubmed-meshheading:8491022-Action Potentials,
pubmed-meshheading:8491022-Animals,
pubmed-meshheading:8491022-Dogs,
pubmed-meshheading:8491022-Electric Stimulation,
pubmed-meshheading:8491022-Heart,
pubmed-meshheading:8491022-Heart Ventricles,
pubmed-meshheading:8491022-Homeostasis,
pubmed-meshheading:8491022-Myocardium,
pubmed-meshheading:8491022-Quinidine,
pubmed-meshheading:8491022-Reaction Time,
pubmed-meshheading:8491022-Tachycardia, Ventricular
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pubmed:year |
1993
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pubmed:articleTitle |
Action potential alternans and irregular dynamics in quinidine-intoxicated ventricular muscle cells. Implications for ventricular proarrhythmia.
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pubmed:affiliation |
Division of Cardiology, Cedars-Sinai Medical Research Institute, Los Angeles, CA.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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