| pubmed-article:8406893 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8406893 | lifeskim:mentions | umls-concept:C0302363 | lld:lifeskim |
| pubmed-article:8406893 | lifeskim:mentions | umls-concept:C0021745 | lld:lifeskim |
| pubmed-article:8406893 | lifeskim:mentions | umls-concept:C0683598 | lld:lifeskim |
| pubmed-article:8406893 | lifeskim:mentions | umls-concept:C0086597 | lld:lifeskim |
| pubmed-article:8406893 | lifeskim:mentions | umls-concept:C0205227 | lld:lifeskim |
| pubmed-article:8406893 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:8406893 | pubmed:dateCreated | 1993-11-24 | lld:pubmed |
| pubmed-article:8406893 | pubmed:abstractText | Depletion of endogenous gamma interferon (IFN-gamma) with anti-IFN-gamma monoclonal antibody resulted in increased numbers of Brucella abortus in the spleen and liver of infected CBA mice. This increase was accompanied by a decrease in splenomegaly and a lower proportion of macrophages in the spleen. Furthermore, treatment of recipient mice with anti-IFN-gamma antibody blocked the adoptive transfer of resistance with immune T cells. Together, the results indicated that endogenous IFN-gamma plays an important role in mediating resistance to primary and secondary Brucella infection. | lld:pubmed |
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| pubmed-article:8406893 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:8406893 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8406893 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8406893 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:8406893 | pubmed:issn | 0019-9567 | lld:pubmed |
| pubmed-article:8406893 | pubmed:author | pubmed-author:CheersCC | lld:pubmed |
| pubmed-article:8406893 | pubmed:author | pubmed-author:ZhanYY | lld:pubmed |
| pubmed-article:8406893 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8406893 | pubmed:volume | 61 | lld:pubmed |
| pubmed-article:8406893 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8406893 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8406893 | pubmed:pagination | 4899-901 | lld:pubmed |
| pubmed-article:8406893 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:8406893 | pubmed:year | 1993 | lld:pubmed |
| pubmed-article:8406893 | pubmed:articleTitle | Endogenous gamma interferon mediates resistance to Brucella abortus infection. | lld:pubmed |
| pubmed-article:8406893 | pubmed:affiliation | Department of Microbiology, University of Melbourne, Parkville, Victoria, Australia. | lld:pubmed |
| pubmed-article:8406893 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8406893 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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