| pubmed-article:8367726 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8367726 | lifeskim:mentions | umls-concept:C0023281 | lld:lifeskim |
| pubmed-article:8367726 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:8367726 | lifeskim:mentions | umls-concept:C0018894 | lld:lifeskim |
| pubmed-article:8367726 | lifeskim:mentions | umls-concept:C1332714 | lld:lifeskim |
| pubmed-article:8367726 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:8367726 | pubmed:issue | 5127 | lld:pubmed |
| pubmed-article:8367726 | pubmed:dateCreated | 1993-10-4 | lld:pubmed |
| pubmed-article:8367726 | pubmed:abstractText | Expression of either the CD4 or CD8 glycoproteins discriminates two functionally distinct lineages of T lymphocytes. A null mutation in the gene encoding CD4 impairs the development of the helper cell lineage that is normally defined by CD4 expression. Infection of CD4-null mice with Leishmania has revealed a population of functional helper T cells that develops despite the absence of CD4. These CD8- alpha beta T cell receptor+ T cells are major histocompatibility complex class II-restricted and produce interferon-gamma when challenged with parasite antigens. These results indicate that T lymphocyte lineage commitment and peripheral function need not depend on the function of CD4. | lld:pubmed |
| pubmed-article:8367726 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8367726 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8367726 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8367726 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8367726 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8367726 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:8367726 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:8367726 | pubmed:author | pubmed-author:LittmanD RDR | lld:pubmed |
| pubmed-article:8367726 | pubmed:author | pubmed-author:LocksleyR MRM | lld:pubmed |
| pubmed-article:8367726 | pubmed:author | pubmed-author:KilleenNN | lld:pubmed |
| pubmed-article:8367726 | pubmed:author | pubmed-author:ReinerS LSL | lld:pubmed |
| pubmed-article:8367726 | pubmed:author | pubmed-author:HataiBB | lld:pubmed |
| pubmed-article:8367726 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8367726 | pubmed:day | 10 | lld:pubmed |
| pubmed-article:8367726 | pubmed:volume | 261 | lld:pubmed |
| pubmed-article:8367726 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8367726 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8367726 | pubmed:pagination | 1448-51 | lld:pubmed |
| pubmed-article:8367726 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:8367726 | pubmed:meshHeading | pubmed-meshheading:8367726-... | lld:pubmed |
| pubmed-article:8367726 | pubmed:year | 1993 | lld:pubmed |
| pubmed-article:8367726 | pubmed:articleTitle | Helper T cells without CD4: control of leishmaniasis in CD4-deficient mice. | lld:pubmed |
| pubmed-article:8367726 | pubmed:affiliation | Department of Medicine, University of California, San Francisco 94143-0654. | lld:pubmed |
| pubmed-article:8367726 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8367726 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:8367726 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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