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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
1
|
pubmed:dateCreated |
1994-2-22
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pubmed:abstractText |
Inhaled nitric oxide (NO), an endothelium-derived relaxing factor, is a selective pulmonary vasodilator. The authors investigated whether inhaled NO decreases pulmonary vascular resistance (PVR) while preserving hypoxic pulmonary vasoconstriction and whether it maintains or improves oxygenation in patients during one-lung ventilation.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
0003-3022
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
80
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
57-62; discussion 27A
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:8291730-Cardiac Surgical Procedures,
pubmed-meshheading:8291730-Humans,
pubmed-meshheading:8291730-Lung,
pubmed-meshheading:8291730-Middle Aged,
pubmed-meshheading:8291730-Nitric Oxide,
pubmed-meshheading:8291730-Oxygen Consumption,
pubmed-meshheading:8291730-Respiration, Artificial,
pubmed-meshheading:8291730-Vascular Resistance
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pubmed:year |
1994
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pubmed:articleTitle |
Inhaled nitric oxide selectively decreases pulmonary vascular resistance without impairing oxygenation during one-lung ventilation in patients undergoing cardiac surgery.
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pubmed:affiliation |
Department of Anesthesiology, University of Virginia Health Sciences Center, Charlottesville.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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