Linked Life Data

8079038

Source:http://linkedlifedata.com/resource/pubmed/id/8079038

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
7-8
pubmed:dateCreated
1994-10-6
pubmed:abstractText
On the basis of an extensive review of the literature and their personal experience, the authors consider that neonatal PILBD should not be regarded as merely a malformative anomaly of the bile excretory system, but as a delayed growth of the pars cystica of the hepatic bud in comparison with the normal growth pattern of the cranial part of the same hepatic bud. This leads to the development of hepatocytes and ductal plate, and these, in turn, are the origin of the perilobular or terminal bile ducts (Hering's ampullae) which eventually fuse with interlobular bile ducts for establishing the continuity of the bile duct system. The authors base this hypothesis on their L.M. and E.M. investigations and the casual observation of two cases in which the well documented bile duct anomaly eventually turned into a normal liver histology.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:status
MEDLINE
pubmed:issn
0034-1193
pubmed:author
pubmed:issnType
Print
pubmed:volume
85
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
375-83
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed:articleTitle
Intrahepatic cholestasis by paucity of interlobular bile ducts in infancy.
pubmed:affiliation
Dipartimento di Medicina Sperimentale, Università La Sapienza, Roma.
pubmed:publicationType
Journal Article, Comparative Study