| pubmed-article:805673 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:805673 | lifeskim:mentions | umls-concept:C0033554 | lld:lifeskim |
| pubmed-article:805673 | lifeskim:mentions | umls-concept:C1753303 | lld:lifeskim |
| pubmed-article:805673 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
| pubmed-article:805673 | lifeskim:mentions | umls-concept:C0205227 | lld:lifeskim |
| pubmed-article:805673 | pubmed:issue | 6 Suppl 1 | lld:pubmed |
| pubmed-article:805673 | pubmed:dateCreated | 1975-9-4 | lld:pubmed |
| pubmed-article:805673 | pubmed:abstractText | Prostaglandins are primarily local or tissue hormones which have their effects at or near the site of release and are metabolized before reaching the arterial circulation. A possible exception is prostaglandin A-2, which has been proposed as a circulating hormone; however, there is no evidence that prostaglandin A-2 is biosynthesized in the mammalian kidney or even in extrarenal tissues. The prostaglandin generated and released in the kidney is predominantly E-2. Some blood vessels also synthesize prostaglandins intramurally, where their local release influences vascular tone and reactivity. Endogenous prostaglandin E-2 production contributes to the regulation of blood pressure by (1) opposing the vasoconstrictor and antidiuretic actions of circulating pressor hormones; (2) braking the release of norepinephrine from vasoconstrictor nerves; and (3) participating in the control extracellular fluid volume through its renal hemodynamic actions. | lld:pubmed |
| pubmed-article:805673 | pubmed:language | eng | lld:pubmed |
| pubmed-article:805673 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:805673 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:805673 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:805673 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:805673 | pubmed:issn | 0009-7330 | lld:pubmed |
| pubmed-article:805673 | pubmed:author | pubmed-author:VaneJ RJR | lld:pubmed |
| pubmed-article:805673 | pubmed:author | pubmed-author:McGiffJ CJC | lld:pubmed |
| pubmed-article:805673 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:805673 | pubmed:volume | 36 | lld:pubmed |
| pubmed-article:805673 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:805673 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:805673 | pubmed:pagination | 68-75 | lld:pubmed |
| pubmed-article:805673 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:805673 | pubmed:year | 1975 | lld:pubmed |
| pubmed-article:805673 | pubmed:articleTitle | Possible contributions of endogenous prostaglandins to the control of blood pressure. | lld:pubmed |
| pubmed-article:805673 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:805673 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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