Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
8 Pt 1
pubmed:dateCreated
1994-9-7
pubmed:abstractText
It has been recently noted that laryngeal paralysis results in a complex alteration of the glottis. The membranous segment of the paralyzed vocal fold is shortened, and, during phonation, patients use hyperfunction to shorten the normal vocal fold to about the same length. Additionally, if the paralyzed vocal fold is not near the midline, the angle between the membranous and cartilaginous segments of the vocal fold is decreased, resulting in a "posterior" gap which cannot be closed by hyperadduction of the normal side. To determine whether arytenoid adduction addresses these problems, videolaryngoscopy was analyzed in 11 patients before and after surgery, and results were compared to patient satisfaction and acoustic and aerodynamic assessment. The posterior gap and glottic competence were improved in all patients, but only 6 had improvement in symptoms. Two had persistent vocal fold bowing but achieved good function after Teflon injection. Three patients, all with paralysis for more than 20 years, had no increase in vocal fold length and very little subjective vocal improvement. Arytenoid adduction is most effective in acute cases. Poor functional results in chronic paralysis are related to failure to achieve vocal fold lengthening, presumably due to soft-tissue contracture.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Aug
pubmed:issn
0023-852X
pubmed:author
pubmed:issnType
Print
pubmed:volume
104
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
965-9
pubmed:dateRevised
2004-11-17
pubmed:meshHeading
pubmed:year
1994
pubmed:articleTitle
Glottic configuration after arytenoid adduction.
pubmed:affiliation
Department of Otolaryngology-Head and Neck Surgery, University of Tennessee, Memphis, College of Medicine 38163.
pubmed:publicationType
Journal Article