| pubmed-article:7877685 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7877685 | lifeskim:mentions | umls-concept:C0018792 | lld:lifeskim |
| pubmed-article:7877685 | lifeskim:mentions | umls-concept:C0033684 | lld:lifeskim |
| pubmed-article:7877685 | lifeskim:mentions | umls-concept:C0439799 | lld:lifeskim |
| pubmed-article:7877685 | pubmed:issue | 6518 | lld:pubmed |
| pubmed-article:7877685 | pubmed:dateCreated | 1995-4-6 | lld:pubmed |
| pubmed-article:7877685 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7877685 | pubmed:abstractText | Heart rate is slowed in part by acetylcholine-dependent activation of a cardiac potassium (K+) channel, IKACh. Activated muscarinic receptors stimulate IKACh via the G-protein beta gamma-subunits. It has been assumed that the inwardly rectifying K(+)-channel gene, GIRK1, alone encodes IKACh. It is now shown that IKACh is a heteromultimer of two distinct inwardly rectifying K(+)-channel subunits, GIRK1 and a newly cloned member of the family, CIR. | lld:pubmed |
| pubmed-article:7877685 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7877685 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7877685 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:7877685 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:7877685 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7877685 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:7877685 | pubmed:issn | 0028-0836 | lld:pubmed |
| pubmed-article:7877685 | pubmed:author | pubmed-author:WickmanKK | lld:pubmed |
| pubmed-article:7877685 | pubmed:author | pubmed-author:ClaphamD EDE | lld:pubmed |
| pubmed-article:7877685 | pubmed:author | pubmed-author:GordonE AEA | lld:pubmed |
| pubmed-article:7877685 | pubmed:author | pubmed-author:Velimirovi?BB | lld:pubmed |
| pubmed-article:7877685 | pubmed:author | pubmed-author:KrapivinskyLL | lld:pubmed |
| pubmed-article:7877685 | pubmed:author | pubmed-author:KrapivinskyGG | lld:pubmed |
| pubmed-article:7877685 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7877685 | pubmed:day | 9 | lld:pubmed |
| pubmed-article:7877685 | pubmed:volume | 374 | lld:pubmed |
| pubmed-article:7877685 | pubmed:geneSymbol | GIRK1 | lld:pubmed |
| pubmed-article:7877685 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7877685 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7877685 | pubmed:pagination | 135-41 | lld:pubmed |
| pubmed-article:7877685 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:7877685 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7877685 | pubmed:articleTitle | The G-protein-gated atrial K+ channel IKACh is a heteromultimer of two inwardly rectifying K(+)-channel proteins. | lld:pubmed |
| pubmed-article:7877685 | pubmed:affiliation | Department of Pharmacology, Mayo Foundation, Rochester, Minnesota 55905. | lld:pubmed |
| pubmed-article:7877685 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7877685 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:7877685 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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