rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
1 Pt 1
|
pubmed:dateCreated |
1995-2-16
|
pubmed:abstractText |
Acquired deficiency in C1-inhibitor (C1-INH) associated with malignancy is often asymptomatic because clinical manifestations are not dependent on a critical complement threshold (in contrast to hereditary C1-INH deficiency). Increased complement consumption involving different kinds of antibodies is the postulated mechanism for this disease, but other factors must play an important role.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Jan
|
pubmed:issn |
0091-6749
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
95
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
124-31
|
pubmed:dateRevised |
2007-11-15
|
pubmed:meshHeading |
pubmed-meshheading:7822653-Aged,
pubmed-meshheading:7822653-Anemia, Hemolytic,
pubmed-meshheading:7822653-Angioedema,
pubmed-meshheading:7822653-Antibodies, Neoplasm,
pubmed-meshheading:7822653-Carcinoma, Signet Ring Cell,
pubmed-meshheading:7822653-Chronic Disease,
pubmed-meshheading:7822653-Complement C1 Inactivator Proteins,
pubmed-meshheading:7822653-Complement System Proteins,
pubmed-meshheading:7822653-Fatal Outcome,
pubmed-meshheading:7822653-Female,
pubmed-meshheading:7822653-Humans,
pubmed-meshheading:7822653-Immunohistochemistry,
pubmed-meshheading:7822653-Neoplasm Recurrence, Local,
pubmed-meshheading:7822653-Stomach,
pubmed-meshheading:7822653-Stomach Neoplasms
|
pubmed:year |
1995
|
pubmed:articleTitle |
Acquired deficiency in C1-inhibitor associated with signet ring cell gastric adenocarcinoma: a probable connection of antitumor-associated antibodies, hemolytic anemia, and complement turnover.
|
pubmed:affiliation |
Department of Internal Medicine, University Hospital (CHUV), Lausanne, Switzerland.
|
pubmed:publicationType |
Journal Article,
Review,
Case Reports
|