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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
3
|
| pubmed:dateCreated |
1995-8-24
|
| pubmed:abstractText |
Taking into account its detailed morphology, an attempt has been made to explain a tendon's ability to transmit force between its muscular origin and skeletal insertion. This ability depends upon the aggregation and binding capacity of the molecular elements of type I collagen and is known to decrease with advancing age. The internal structure of a tendon is modified both at the muscle-tendon junction and near its attachment to bone. The very low elasticity of tendinous tissue (about 3% to 8%) is due partly to the texture (pseudo-elasticity) and partly to the molecular composition. The so-called degenerative changes in a tendon begin with deterioration of the internal vascular network and are obviously--so far as its failing capacity is concerned--both initiated and accelerated by the degree of over-loading.
|
| pubmed:language |
ger
|
| pubmed:journal | |
| pubmed:citationSubset |
IM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:month |
Jun
|
| pubmed:issn |
0085-4530
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:volume |
24
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
180-6
|
| pubmed:dateRevised |
2006-11-15
|
| pubmed:meshHeading | |
| pubmed:year |
1995
|
| pubmed:articleTitle |
[Anatomy and pathology of tendons].
|
| pubmed:affiliation |
Anatomische Anstalt (Lehrstuhl I), Ludwig-Maximilians-Universität München.
|
| pubmed:publicationType |
Journal Article,
English Abstract,
Review,
Research Support, Non-U.S. Gov't
|