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rdf:type |
|
|
lifeskim:mentions |
|
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pubmed:issue |
11
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pubmed:dateCreated |
1995-11-30
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|
pubmed:abstractText |
Bartonella henselae expresses pili phenotypically similar to type 4 pili. B. henselae pilus expression undergoes phase variation with multiple passages. Low-passage-number, piliated B. henselae adhered to and invaded HEp-2 cells to a greater extent than did multiply passaged B. henselae with reduced pilus expression. Pili may be a pathogenic determinant for Bartonella species.
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pubmed:grant |
|
|
pubmed:commentsCorrections |
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pubmed:language |
eng
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|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Nov
|
|
pubmed:issn |
0019-9567
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|
pubmed:author |
|
|
pubmed:issnType |
Print
|
|
pubmed:volume |
63
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
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pubmed:pagination |
4553-6
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|
pubmed:dateRevised |
2009-11-18
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|
pubmed:meshHeading |
|
|
pubmed:year |
1995
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|
pubmed:articleTitle |
Bartonella henselae and Bartonella quintana adherence to and entry into cultured human epithelial cells.
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|
pubmed:affiliation |
Department of Medicine, Stanford University, California 94305, USA.
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|
pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
|
