| pubmed-article:7579194 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7579194 | lifeskim:mentions | umls-concept:C0022688 | lld:lifeskim |
| pubmed-article:7579194 | lifeskim:mentions | umls-concept:C0037083 | lld:lifeskim |
| pubmed-article:7579194 | lifeskim:mentions | umls-concept:C0205160 | lld:lifeskim |
| pubmed-article:7579194 | lifeskim:mentions | umls-concept:C1446409 | lld:lifeskim |
| pubmed-article:7579194 | lifeskim:mentions | umls-concept:C1369008 | lld:lifeskim |
| pubmed-article:7579194 | lifeskim:mentions | umls-concept:C1710082 | lld:lifeskim |
| pubmed-article:7579194 | lifeskim:mentions | umls-concept:C0591833 | lld:lifeskim |
| pubmed-article:7579194 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:7579194 | pubmed:dateCreated | 1995-12-26 | lld:pubmed |
| pubmed-article:7579194 | pubmed:abstractText | Murine NK cells can reject allogenic or parental-strain bone marrow cells (BMC) in vivo and can lyse T lymphoblasts in vitro. The 'missing self' hypothesis states that absence or presence of 'negative signals' from target cell class I antigens (Ag) to NK receptors determines whether or not lysis occurs. Indeed, lysis of parental-strain blasts by purified F1 NK cell subsets occurred only in the presence of anti-receptor antibodies. Evidence for 'positive signaling' to NK cells by class I Ag includes rejection of D8 (Dd) transgene to B6) BMC by B6 hosts. The outcome of other BMC transplants contradict the missing self idea, because donors with identical class I Ag differ in compatibility with certain hosts. Perhaps class I Ag-NK cell receptor interactions dominate over other target-NK cell interactions. These interactions are usually 'negative' but can be 'positive'. | lld:pubmed |
| pubmed-article:7579194 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7579194 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7579194 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7579194 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7579194 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7579194 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:7579194 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7579194 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:7579194 | pubmed:issn | 1044-5323 | lld:pubmed |
| pubmed-article:7579194 | pubmed:author | pubmed-author:YuY YYY | lld:pubmed |
| pubmed-article:7579194 | pubmed:author | pubmed-author:BennettMM | lld:pubmed |
| pubmed-article:7579194 | pubmed:author | pubmed-author:LindahlK FKF | lld:pubmed |
| pubmed-article:7579194 | pubmed:author | pubmed-author:KumarVV | lld:pubmed |
| pubmed-article:7579194 | pubmed:author | pubmed-author:MathewP APA | lld:pubmed |
| pubmed-article:7579194 | pubmed:author | pubmed-author:RembeckiR MRM | lld:pubmed |
| pubmed-article:7579194 | pubmed:author | pubmed-author:StonemanEE | lld:pubmed |
| pubmed-article:7579194 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7579194 | pubmed:volume | 7 | lld:pubmed |
| pubmed-article:7579194 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7579194 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7579194 | pubmed:pagination | 121-7 | lld:pubmed |
| pubmed-article:7579194 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
| pubmed-article:7579194 | pubmed:meshHeading | pubmed-meshheading:7579194-... | lld:pubmed |
| pubmed-article:7579194 | pubmed:meshHeading | pubmed-meshheading:7579194-... | lld:pubmed |
| pubmed-article:7579194 | pubmed:meshHeading | pubmed-meshheading:7579194-... | lld:pubmed |
| pubmed-article:7579194 | pubmed:meshHeading | pubmed-meshheading:7579194-... | lld:pubmed |
| pubmed-article:7579194 | pubmed:meshHeading | pubmed-meshheading:7579194-... | lld:pubmed |
| pubmed-article:7579194 | pubmed:meshHeading | pubmed-meshheading:7579194-... | lld:pubmed |
| pubmed-article:7579194 | pubmed:meshHeading | pubmed-meshheading:7579194-... | lld:pubmed |
| pubmed-article:7579194 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7579194 | pubmed:articleTitle | Hybrid resistance: 'negative' and 'positive' signaling of murine natural killer cells. | lld:pubmed |
| pubmed-article:7579194 | pubmed:affiliation | Department of Pathology, University of Texas Southwestern Medical Center at Dallas, USA. | lld:pubmed |
| pubmed-article:7579194 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7579194 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:7579194 | pubmed:publicationType | Review | lld:pubmed |
| pubmed-article:7579194 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:7579194 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:7579194 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:7579194 | lld:pubmed |
