| pubmed-article:7545940 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7545940 | lifeskim:mentions | umls-concept:C0014834 | lld:lifeskim |
| pubmed-article:7545940 | lifeskim:mentions | umls-concept:C0019892 | lld:lifeskim |
| pubmed-article:7545940 | lifeskim:mentions | umls-concept:C0038187 | lld:lifeskim |
| pubmed-article:7545940 | lifeskim:mentions | umls-concept:C0086982 | lld:lifeskim |
| pubmed-article:7545940 | pubmed:issue | 5171 | lld:pubmed |
| pubmed-article:7545940 | pubmed:dateCreated | 1994-8-15 | lld:pubmed |
| pubmed-article:7545940 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7545940 | pubmed:abstractText | When nutrients become limiting, many bacteria differentiate and become resistant to environmental stresses. For Escherichia coli, this process is mediated by the sigma s subunit of RNA polymerase. Expression of sigma s was induced by homoserine lactone, a metabolite synthesized from intermediates in threonine biosynthesis. Homoserine lactone-dependent synthesis of sigma s was prevented by overexpression of a newly identified protein, RspA. The function of homoserine lactone derivatives in many cell density-dependent phenomena and the similarity of RspA to a Streptomyces ambofaciens protein suggest that synthesis of homoserine lactone may be a general signal of starvation. | lld:pubmed |
| pubmed-article:7545940 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7545940 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7545940 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:7545940 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:7545940 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7545940 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:7545940 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:7545940 | pubmed:author | pubmed-author:KolterRR | lld:pubmed |
| pubmed-article:7545940 | pubmed:author | pubmed-author:HuismanG WGW | lld:pubmed |
| pubmed-article:7545940 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7545940 | pubmed:day | 22 | lld:pubmed |
| pubmed-article:7545940 | pubmed:volume | 265 | lld:pubmed |
| pubmed-article:7545940 | pubmed:geneSymbol | rspA | lld:pubmed |
| pubmed-article:7545940 | pubmed:geneSymbol | rpoS | lld:pubmed |
| pubmed-article:7545940 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7545940 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7545940 | pubmed:pagination | 537-9 | lld:pubmed |
| pubmed-article:7545940 | pubmed:dateRevised | 2007-3-19 | lld:pubmed |
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| pubmed-article:7545940 | pubmed:year | 1994 | lld:pubmed |
| pubmed-article:7545940 | pubmed:articleTitle | Sensing starvation: a homoserine lactone--dependent signaling pathway in Escherichia coli. | lld:pubmed |
| pubmed-article:7545940 | pubmed:affiliation | Department of Microbiology and Molecular Genetics, Harvard Medical School, Boston, MA 02115. | lld:pubmed |
| pubmed-article:7545940 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7545940 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:7545940 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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