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pubmed-article:6629414pubmed:abstractTextDecreased lung clearance of Staphylococcus aureus has been reported in mice homozygous for the cribriform degeneration (cri) autosomal recessive mutation. In the present study, the phagocytic capacities of alveolar and peritoneal macrophages were quantitated by applying kinetics of the first order reaction criteria. The characteristics of the pulmonary and peritoneal mononuclear cell populations from mutant and control mice were indistinguishable. The kinetic assays revealed decreased phagocytosis work in both alveolar and peritoneal macrophages from cri/cri mice. The results lend support to this mutation as a possible model system to study the early stages of lung disease physiopathology in cystic fibrosis.lld:pubmed
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pubmed-article:6629414pubmed:authorpubmed-author:PivettaO HOHlld:pubmed
pubmed-article:6629414pubmed:authorpubmed-author:SordelliD ODOlld:pubmed
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pubmed-article:6629414pubmed:volume12lld:pubmed
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pubmed-article:6629414pubmed:pagination375-85lld:pubmed
pubmed-article:6629414pubmed:dateRevised2006-11-15lld:pubmed
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pubmed-article:6629414pubmed:year1983lld:pubmed
pubmed-article:6629414pubmed:articleTitleDefective mononuclear phagocytic function in mice homozygous for the cribriform degeneration autosomic recessive mutation.lld:pubmed
pubmed-article:6629414pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:6629414pubmed:publicationTypeComparative Studylld:pubmed
pubmed-article:6629414pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed