| pubmed-article:6583679 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:6583679 | lifeskim:mentions | umls-concept:C0010453 | lld:lifeskim |
| pubmed-article:6583679 | lifeskim:mentions | umls-concept:C0014597 | lld:lifeskim |
| pubmed-article:6583679 | lifeskim:mentions | umls-concept:C0022646 | lld:lifeskim |
| pubmed-article:6583679 | lifeskim:mentions | umls-concept:C0007595 | lld:lifeskim |
| pubmed-article:6583679 | lifeskim:mentions | umls-concept:C0033684 | lld:lifeskim |
| pubmed-article:6583679 | lifeskim:mentions | umls-concept:C0815327 | lld:lifeskim |
| pubmed-article:6583679 | lifeskim:mentions | umls-concept:C2348693 | lld:lifeskim |
| pubmed-article:6583679 | lifeskim:mentions | umls-concept:C0597484 | lld:lifeskim |
| pubmed-article:6583679 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:6583679 | pubmed:dateCreated | 1984-4-12 | lld:pubmed |
| pubmed-article:6583679 | pubmed:abstractText | Proliferation of confluent kidney epithelial cell cultures (BSC-1 line) is inhibited by a protein (Mr approximately equal to 24,000) that is secreted by the cells. The mechanism of action of this growth inhibitor was sought by studying its effect on net Na+ flux because increased availability of Na+ in the culture medium had been shown to stimulate cell growth. The increase in cell Na+ content observed during stimulation of the growth after a medium change was attenuated in the presence of the purified inhibitor. Inhibition of both cell Na+ accumulation and growth in the presence of the protein was reversed completely by addition of NaCl to the medium. These results suggest that control of net Na+ flux and growth in kidney epithelial cells could be mediated, at least in part, by a secreted cellular protein. | lld:pubmed |
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| pubmed-article:6583679 | pubmed:language | eng | lld:pubmed |
| pubmed-article:6583679 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6583679 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:6583679 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6583679 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:6583679 | pubmed:issn | 0027-8424 | lld:pubmed |
| pubmed-article:6583679 | pubmed:author | pubmed-author:TobackF GFG | lld:pubmed |
| pubmed-article:6583679 | pubmed:author | pubmed-author:HolleyR WRW | lld:pubmed |
| pubmed-article:6583679 | pubmed:author | pubmed-author:Walsh-ReitzM... | lld:pubmed |
| pubmed-article:6583679 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6583679 | pubmed:volume | 81 | lld:pubmed |
| pubmed-article:6583679 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6583679 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6583679 | pubmed:pagination | 793-6 | lld:pubmed |
| pubmed-article:6583679 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:6583679 | pubmed:meshHeading | pubmed-meshheading:6583679-... | lld:pubmed |
| pubmed-article:6583679 | pubmed:year | 1984 | lld:pubmed |
| pubmed-article:6583679 | pubmed:articleTitle | Cell growth and net Na+ flux are inhibited by a protein produced by kidney epithelial cells in culture. | lld:pubmed |
| pubmed-article:6583679 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:6583679 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:6583679 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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