Linked Life Data

6262136

Source:http://linkedlifedata.com/resource/pubmed/id/6262136

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
7
pubmed:dateCreated
1981-7-23
pubmed:abstractText
Acute and chronic administration of ethanol has multiple effects on several neurotransmitters in the basal ganglia. Dopamine is the transmitter predominantly affected. Acceleration of dopaminergic activity is observed at low doses of ethanol. However, at high doses the reverse is observed. During the ethanol withdrawal syndrome that develops after chronic treatment, dopaminergic responses are reduced, whether from presynaptic or postsynaptic origins. Evidence also indicates that cholinergic and GABAergic processes may be implicated in these actions of ethanol. Ethanol apparently induces a variety of alterations in neurotransmitter function as a result of its disruption of membrane structure and associated electric properties.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
0014-9446
pubmed:author
pubmed:issnType
Print
pubmed:day
15
pubmed:volume
40
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2077-81
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed:year
1981
pubmed:articleTitle
Neurotransmitter function in the basal ganglia after acute and chronic ethanol treatment.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, Non-P.H.S.