pubmed-article:3725199 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:3725199 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
pubmed-article:3725199 | lifeskim:mentions | umls-concept:C0228174 | lld:lifeskim |
pubmed-article:3725199 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
pubmed-article:3725199 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
pubmed-article:3725199 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:3725199 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
pubmed-article:3725199 | lifeskim:mentions | umls-concept:C1515655 | lld:lifeskim |
pubmed-article:3725199 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:3725199 | pubmed:dateCreated | 1986-8-13 | lld:pubmed |
pubmed-article:3725199 | pubmed:abstractText | The effects of gamma-aminobutyric acid transaminase (GABA-T) inhibitors on rat endothelial GABA-T activity has been studied utilizing a modified tetrazolium salt histochemical technique. Blue-diformazan staining of cerebrovascular and parenchymal sites of GABA catabolism in normal brain was almost completely prevented in unfixed frozen brain sections from rats subjected to 'in vivo' GABA-T inhibition using aminooxyacetic acid or gabaculine. These results provide histochemical evidence for the efficacy of using systemically administered GABA-T inhibitors to inhibit endothelial GABA-T activity. | lld:pubmed |
pubmed-article:3725199 | pubmed:language | eng | lld:pubmed |
pubmed-article:3725199 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3725199 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:3725199 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3725199 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3725199 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3725199 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3725199 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3725199 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:3725199 | pubmed:month | Jun | lld:pubmed |
pubmed-article:3725199 | pubmed:issn | 0304-3940 | lld:pubmed |
pubmed-article:3725199 | pubmed:author | pubmed-author:KrantisAA | lld:pubmed |
pubmed-article:3725199 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:3725199 | pubmed:day | 6 | lld:pubmed |
pubmed-article:3725199 | pubmed:volume | 67 | lld:pubmed |
pubmed-article:3725199 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:3725199 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:3725199 | pubmed:pagination | 48-52 | lld:pubmed |
pubmed-article:3725199 | pubmed:dateRevised | 2003-11-14 | lld:pubmed |
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pubmed-article:3725199 | pubmed:year | 1986 | lld:pubmed |
pubmed-article:3725199 | pubmed:articleTitle | Cerebral endothelial GABA-T activity: effects of in vivo GABA-T inhibition. | lld:pubmed |
pubmed-article:3725199 | pubmed:publicationType | Journal Article | lld:pubmed |