rdf:type |
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lifeskim:mentions |
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pubmed:dateCreated |
2011-7-4
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pubmed:abstractText |
Oxidative stress contributes significantly to the development of secondary brain injury after intracerebral hemorrhage (ICH). It has been previously demonstrated that hydrogen gas can decrease oxidative stress by scavenging reactive oxygen species. We hypothesized that hydrogen therapy will reduce brain oxidative stress in mice after ICH and thereby will lead to reduced brain edema and improved neurological outcomes.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
0065-1419
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
111
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
179-83
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pubmed:meshHeading |
pubmed-meshheading:21725752-Administration, Inhalation,
pubmed-meshheading:21725752-Analysis of Variance,
pubmed-meshheading:21725752-Animals,
pubmed-meshheading:21725752-Brain,
pubmed-meshheading:21725752-Brain Edema,
pubmed-meshheading:21725752-Cerebral Hemorrhage,
pubmed-meshheading:21725752-Collagenases,
pubmed-meshheading:21725752-Disease Models, Animal,
pubmed-meshheading:21725752-Functional Laterality,
pubmed-meshheading:21725752-Hydrogen,
pubmed-meshheading:21725752-Male,
pubmed-meshheading:21725752-Mice,
pubmed-meshheading:21725752-Motor Skills,
pubmed-meshheading:21725752-Nervous System Diseases,
pubmed-meshheading:21725752-Neurologic Examination,
pubmed-meshheading:21725752-Neuroprotective Agents,
pubmed-meshheading:21725752-Time Factors
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pubmed:year |
2011
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pubmed:articleTitle |
Hydrogen inhalation is neuroprotective and improves functional outcomes in mice after intracerebral hemorrhage.
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pubmed:affiliation |
Department of Physiology and Pharmacology, Loma Linda University, School of Medicine, Loma Linda, CA 92350, USA.
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pubmed:publicationType |
Journal Article,
Research Support, N.I.H., Extramural
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