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pubmed-article:21551411pubmed:abstractTextRPE65 gene knockout (Rpe65?/?) mice showed abolished isomerohydrolase activity in the visual cycle and were considered a model for vitamin A deficiency in the retina. The purpose of this study was to compare the retinal phenotypes between vitamin A-deficient (VAD) mice and Rpe65?/? mice under normal diet.lld:pubmed
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pubmed-article:21551411pubmed:dateRevised2011-9-30lld:pubmed
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pubmed-article:21551411pubmed:articleTitleComparison of ocular pathologies in vitamin A-deficient mice and RPE65 gene knockout mice.lld:pubmed
pubmed-article:21551411pubmed:affiliationDepartment of Physiology, Department of Medicine and Harold Hamm Oklahoma Diabetes Center, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA.lld:pubmed
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