Linked Life Data

21270276

Source:http://linkedlifedata.com/resource/pubmed/id/21270276

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2011-1-28
pubmed:abstractText
To determine the mechanisms by which blockade of adenosine A(2B) receptors (A(2B)Rs) reduces insulin resistance.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1939-327X
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
60
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
669-79
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed-meshheading:21270276-Adenosine-5'-(N-ethylcarboxamide), pubmed-meshheading:21270276-Animals, pubmed-meshheading:21270276-Biological Markers, pubmed-meshheading:21270276-Blood Glucose, pubmed-meshheading:21270276-C-Reactive Protein, pubmed-meshheading:21270276-Cells, Cultured, pubmed-meshheading:21270276-Diabetes Mellitus, Type 2, pubmed-meshheading:21270276-Glucose Clamp Technique, pubmed-meshheading:21270276-Glucose Tolerance Test, pubmed-meshheading:21270276-Humans, pubmed-meshheading:21270276-Inflammation, pubmed-meshheading:21270276-Insulin, pubmed-meshheading:21270276-Insulin Resistance, pubmed-meshheading:21270276-Interleukin-6, pubmed-meshheading:21270276-Liver, pubmed-meshheading:21270276-Macrophages, pubmed-meshheading:21270276-Mice, pubmed-meshheading:21270276-Mice, Transgenic, pubmed-meshheading:21270276-Polymorphism, Single Nucleotide, pubmed-meshheading:21270276-Receptor, Adenosine A2B, pubmed-meshheading:21270276-Reverse Transcriptase Polymerase Chain Reaction
pubmed:year
2011
pubmed:articleTitle
Links between insulin resistance, adenosine A2B receptors, and inflammatory markers in mice and humans.
pubmed:affiliation
Cardiovascular Research Center, University of Virginia, Charlottesville, Virginia, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, Non-P.H.S., Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural