Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2011-1-28
pubmed:abstractText
Previous findings demonstrate that enhanced expression of the forkhead transcription factor Foxc2 in adipose tissue leads to a lean and insulin-sensitive phenotype. These findings prompted us to further investigate the role of Foxc2 in the regulation of genes of fundamental importance for metabolism and mitochondrial function.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1939-327X
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
60
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
427-35
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed-meshheading:21270254-3T3 Cells, pubmed-meshheading:21270254-Adipocytes, pubmed-meshheading:21270254-Adipose Tissue, pubmed-meshheading:21270254-Aminoimidazole Carboxamide, pubmed-meshheading:21270254-Analysis of Variance, pubmed-meshheading:21270254-Animals, pubmed-meshheading:21270254-Blotting, Western, pubmed-meshheading:21270254-Cells, Cultured, pubmed-meshheading:21270254-Fatty Acids, pubmed-meshheading:21270254-Female, pubmed-meshheading:21270254-Forkhead Transcription Factors, pubmed-meshheading:21270254-Gene Expression Regulation, pubmed-meshheading:21270254-HEK293 Cells, pubmed-meshheading:21270254-Humans, pubmed-meshheading:21270254-Hypoglycemic Agents, pubmed-meshheading:21270254-Insulin, pubmed-meshheading:21270254-Male, pubmed-meshheading:21270254-Mice, pubmed-meshheading:21270254-Microscopy, Electron, pubmed-meshheading:21270254-Mitochondria, pubmed-meshheading:21270254-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:21270254-Ribonucleotides, pubmed-meshheading:21270254-Transfection
pubmed:year
2011
pubmed:articleTitle
The adipocyte-expressed forkhead transcription factor Foxc2 regulates metabolism through altered mitochondrial function.
pubmed:affiliation
Department of Medical and Clinical Genetics, Institute of Biomedicine, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't