Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2011-1-28
pubmed:abstractText
The hypothesis that high-fat (HF) feeding causes skeletal muscle extracellular matrix (ECM) remodeling in C57BL/6J mice and that this remodeling contributes to diet-induced muscle insulin resistance (IR) through the collagen receptor integrin ?(2)?(1) was tested.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1939-327X
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
60
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
416-26
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed-meshheading:21270253-Analysis of Variance, pubmed-meshheading:21270253-Animals, pubmed-meshheading:21270253-Blotting, Western, pubmed-meshheading:21270253-Collagen Type III, pubmed-meshheading:21270253-Collagen Type V, pubmed-meshheading:21270253-Diet, pubmed-meshheading:21270253-Extracellular Matrix, pubmed-meshheading:21270253-Glucose Clamp Technique, pubmed-meshheading:21270253-Immunohistochemistry, pubmed-meshheading:21270253-Insulin, pubmed-meshheading:21270253-Insulin Resistance, pubmed-meshheading:21270253-Integrin alpha2beta1, pubmed-meshheading:21270253-Matrix Metalloproteinase 9, pubmed-meshheading:21270253-Mice, pubmed-meshheading:21270253-Mice, Knockout, pubmed-meshheading:21270253-Muscle, Skeletal, pubmed-meshheading:21270253-Phosphodiesterase 5 Inhibitors, pubmed-meshheading:21270253-Piperazines, pubmed-meshheading:21270253-Purines, pubmed-meshheading:21270253-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:21270253-Sulfones
pubmed:year
2011
pubmed:articleTitle
Diet-induced muscle insulin resistance is associated with extracellular matrix remodeling and interaction with integrin alpha2beta1 in mice.
pubmed:affiliation
Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, Tennessee, USA. li.kang@vanderbilt.edu
pubmed:publicationType
Journal Article, Research Support, N.I.H., Extramural