| pubmed-article:2124708 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2124708 | lifeskim:mentions | umls-concept:C0008633 | lld:lifeskim |
| pubmed-article:2124708 | lifeskim:mentions | umls-concept:C0033684 | lld:lifeskim |
| pubmed-article:2124708 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:2124708 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:2124708 | lifeskim:mentions | umls-concept:C0013139 | lld:lifeskim |
| pubmed-article:2124708 | lifeskim:mentions | umls-concept:C0301625 | lld:lifeskim |
| pubmed-article:2124708 | pubmed:issue | 24 | lld:pubmed |
| pubmed-article:2124708 | pubmed:dateCreated | 1991-2-7 | lld:pubmed |
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| pubmed-article:2124708 | pubmed:abstractText | We report here that a point mutation in the gene which encodes the heterochromatin-specific nonhistone chromosomal protein HP-1 in Drosophila melanogaster is associated with dominant suppression of position-effect variegation. The mutation, a G-to-A transition at the first nucleotide of the last intron, causes missplicing of the HP-1 mRNA. This suggests that heterochromatin-specific proteins play a central role in the gene suppression associated with heterochromatic position effects. | lld:pubmed |
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| pubmed-article:2124708 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2124708 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2124708 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2124708 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2124708 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:2124708 | pubmed:issn | 0027-8424 | lld:pubmed |
| pubmed-article:2124708 | pubmed:author | pubmed-author:ElginS CSC | lld:pubmed |
| pubmed-article:2124708 | pubmed:author | pubmed-author:JamesT CTC | lld:pubmed |
| pubmed-article:2124708 | pubmed:author | pubmed-author:EissenbergJ... | lld:pubmed |
| pubmed-article:2124708 | pubmed:author | pubmed-author:HartnettTT | lld:pubmed |
| pubmed-article:2124708 | pubmed:author | pubmed-author:LaceE BEB | lld:pubmed |
| pubmed-article:2124708 | pubmed:author | pubmed-author:Foster-Hartne... | lld:pubmed |
| pubmed-article:2124708 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2124708 | pubmed:volume | 87 | lld:pubmed |
| pubmed-article:2124708 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2124708 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2124708 | pubmed:pagination | 9923-7 | lld:pubmed |
| pubmed-article:2124708 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2124708 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2124708 | pubmed:articleTitle | Mutation in a heterochromatin-specific chromosomal protein is associated with suppression of position-effect variegation in Drosophila melanogaster. | lld:pubmed |
| pubmed-article:2124708 | pubmed:affiliation | E. A. Doisy Department of Biochemistry and Molecular Biology, St. Louis University School of Medicine, MO 63104. | lld:pubmed |
| pubmed-article:2124708 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2124708 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:2124708 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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