| pubmed-article:2108318 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2108318 | lifeskim:mentions | umls-concept:C0085828 | lld:lifeskim |
| pubmed-article:2108318 | lifeskim:mentions | umls-concept:C1366557 | lld:lifeskim |
| pubmed-article:2108318 | lifeskim:mentions | umls-concept:C1704256 | lld:lifeskim |
| pubmed-article:2108318 | lifeskim:mentions | umls-concept:C0439855 | lld:lifeskim |
| pubmed-article:2108318 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:2108318 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:2108318 | pubmed:dateCreated | 1990-5-2 | lld:pubmed |
| pubmed-article:2108318 | pubmed:abstractText | The multifunctional actions of transforming growth factor beta 1 (TGF-beta 1) indicate that it has a pivotal control function in many physiological and pathological processes. An important property of TGF-beta 1 is its ability to activate its own mRNA expression and thereby increase its own secretion. Two distinct regions of the promoter of the TGF-beta 1 gene are responsive to autoregulation: one 5' to the upstream transcriptional start site and another located between the two major start sites. In both promoter regions, autoinduction is mediated by binding of the AP-1 (Jun-Fos) complex. An important contribution to this positive regulation is the autoactivation of c-jun transcription by AP-1. Cotransfection of antisense c-jun or antisense c-fos expression vectors prevents TGF-beta 1 autoinduction. These results demonstrate that both components of the AP-1 complex are required for TGF-beta 1 autoinduction. Induction of jun expression by TGF-beta 1, as well as jun autoinduction, may amplify the action of TGF-beta 1 during normal development and oncogenesis. | lld:pubmed |
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| pubmed-article:2108318 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:2108318 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2108318 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2108318 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:2108318 | pubmed:issn | 0270-7306 | lld:pubmed |
| pubmed-article:2108318 | pubmed:author | pubmed-author:HattoriKK | lld:pubmed |
| pubmed-article:2108318 | pubmed:author | pubmed-author:KimS JSJ | lld:pubmed |
| pubmed-article:2108318 | pubmed:author | pubmed-author:SpornM BMB | lld:pubmed |
| pubmed-article:2108318 | pubmed:author | pubmed-author:RobertsA BAB | lld:pubmed |
| pubmed-article:2108318 | pubmed:author | pubmed-author:KarinMM | lld:pubmed |
| pubmed-article:2108318 | pubmed:author | pubmed-author:KimK YKY | lld:pubmed |
| pubmed-article:2108318 | pubmed:author | pubmed-author:AngerVV | lld:pubmed |
| pubmed-article:2108318 | pubmed:author | pubmed-author:LafyatisRR | lld:pubmed |
| pubmed-article:2108318 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2108318 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:2108318 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2108318 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2108318 | pubmed:pagination | 1492-7 | lld:pubmed |
| pubmed-article:2108318 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:2108318 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2108318 | pubmed:articleTitle | Autoinduction of transforming growth factor beta 1 is mediated by the AP-1 complex. | lld:pubmed |
| pubmed-article:2108318 | pubmed:affiliation | Laboratory of Chemoprevention, National Cancer Institute, Bethesda, Maryland 20892. | lld:pubmed |
| pubmed-article:2108318 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2108318 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:2108318 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:2108318 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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