rdf:type |
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lifeskim:mentions |
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pubmed:dateCreated |
1991-5-24
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pubmed:abstractText |
The effects of stable PGI analogue TTC-909 on CBF and glucose metabolism was studied in the chronic stage of cerebral ischaemia produced by occluding the distal MCA in SHRSP. Administration of TTC-909 (100 ng/kg/day during 7 days) prevented the development of ischaemic oedema and improved secondary metabolic derangement coupled to flow in postischaemic tissues, particularly in the ischaemic rim.
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
0065-1419
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pubmed:author |
|
pubmed:issnType |
Print
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pubmed:volume |
51
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
242-4
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pubmed:dateRevised |
2009-11-11
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pubmed:meshHeading |
pubmed-meshheading:2089907-Animals,
pubmed-meshheading:2089907-Brain,
pubmed-meshheading:2089907-Brain Ischemia,
pubmed-meshheading:2089907-Cerebrovascular Circulation,
pubmed-meshheading:2089907-Cerebrovascular Disorders,
pubmed-meshheading:2089907-Disease Susceptibility,
pubmed-meshheading:2089907-Epoprostenol,
pubmed-meshheading:2089907-Glucose,
pubmed-meshheading:2089907-Male,
pubmed-meshheading:2089907-Rats,
pubmed-meshheading:2089907-Rats, Inbred SHR,
pubmed-meshheading:2089907-Tissue Distribution
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pubmed:year |
1990
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pubmed:articleTitle |
Post-ischaemic treatment with the prostacycline analogue TTC-909 reduces ischaemic brain injury.
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pubmed:affiliation |
Department of Neurosurgery, National Defense Medical College, Saitama, Japan.
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pubmed:publicationType |
Journal Article
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