pubmed-article:2087996 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:2087996 | lifeskim:mentions | umls-concept:C0085979 | lld:lifeskim |
pubmed-article:2087996 | lifeskim:mentions | umls-concept:C0030352 | lld:lifeskim |
pubmed-article:2087996 | lifeskim:mentions | umls-concept:C0025853 | lld:lifeskim |
pubmed-article:2087996 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:2087996 | lifeskim:mentions | umls-concept:C0001272 | lld:lifeskim |
pubmed-article:2087996 | lifeskim:mentions | umls-concept:C1710637 | lld:lifeskim |
pubmed-article:2087996 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:2087996 | pubmed:dateCreated | 1991-5-23 | lld:pubmed |
pubmed-article:2087996 | pubmed:abstractText | The effects of metoclopramide (MCP) on Vmax of action potentials of guinea pig papillary muscles were studied with intracellular microelectrodes. MCP 10-550 mumol/L abbreviated the action potential duration at 90% repolarization (APD90) and decreased the maximal rate of rise of action potential (Vmax) dose-dependently. MCP caused rate-dependent and voltage-dependent reductions of Vmax and retardation of the recovery of Vmax. The results present the possible causes of depressant effect of MCP on Na channel. | lld:pubmed |
pubmed-article:2087996 | pubmed:language | chi | lld:pubmed |
pubmed-article:2087996 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2087996 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:2087996 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2087996 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2087996 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:2087996 | pubmed:month | May | lld:pubmed |
pubmed-article:2087996 | pubmed:issn | 0253-9756 | lld:pubmed |
pubmed-article:2087996 | pubmed:author | pubmed-author:TanTT | lld:pubmed |
pubmed-article:2087996 | pubmed:author | pubmed-author:ZhaoG SGS | lld:pubmed |
pubmed-article:2087996 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:2087996 | pubmed:volume | 11 | lld:pubmed |
pubmed-article:2087996 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:2087996 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:2087996 | pubmed:pagination | 229-31 | lld:pubmed |
pubmed-article:2087996 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
pubmed-article:2087996 | pubmed:meshHeading | pubmed-meshheading:2087996-... | lld:pubmed |
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pubmed-article:2087996 | pubmed:year | 1990 | lld:pubmed |
pubmed-article:2087996 | pubmed:articleTitle | [Effects of metoclopramide on Vmax of action potentials in guinea pig papillary muscles]. | lld:pubmed |
pubmed-article:2087996 | pubmed:affiliation | Department of Pharmacology, Xi-an Medical University, China. | lld:pubmed |
pubmed-article:2087996 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:2087996 | pubmed:publicationType | English Abstract | lld:pubmed |