Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
18
pubmed:dateCreated
2010-5-11
pubmed:abstractText
Members of the tumor necrosis factor superfamily, such as tumor necrosis factor-alpha, potently promote atherogenesis in mice and humans. Tumor necrosis factor receptor-associated factors (TRAFs) are cytoplasmic adaptor proteins for this group of cytokines.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
11
pubmed:volume
121
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2033-44
pubmed:dateRevised
2011-3-1
pubmed:meshHeading
pubmed-meshheading:20421522-Actins, pubmed-meshheading:20421522-Acute Coronary Syndrome, pubmed-meshheading:20421522-Aged, pubmed-meshheading:20421522-Animals, pubmed-meshheading:20421522-Apoptosis, pubmed-meshheading:20421522-Atherosclerosis, pubmed-meshheading:20421522-Bone Marrow Cells, pubmed-meshheading:20421522-Cell Adhesion, pubmed-meshheading:20421522-Cell Differentiation, pubmed-meshheading:20421522-Cell Movement, pubmed-meshheading:20421522-Cells, Cultured, pubmed-meshheading:20421522-Chemokines, pubmed-meshheading:20421522-Endothelial Cells, pubmed-meshheading:20421522-Female, pubmed-meshheading:20421522-Humans, pubmed-meshheading:20421522-Interleukin-6, pubmed-meshheading:20421522-Macrophages, pubmed-meshheading:20421522-Male, pubmed-meshheading:20421522-Mice, pubmed-meshheading:20421522-Mice, Mutant Strains, pubmed-meshheading:20421522-Middle Aged, pubmed-meshheading:20421522-Receptors, Chemokine, pubmed-meshheading:20421522-Receptors, LDL, pubmed-meshheading:20421522-TNF Receptor-Associated Factor 1, pubmed-meshheading:20421522-Vasculitis
pubmed:year
2010
pubmed:articleTitle
Tumor necrosis factor receptor-associated factor 1 (TRAF1) deficiency attenuates atherosclerosis in mice by impairing monocyte recruitment to the vessel wall.
pubmed:affiliation
Department of Cardiology, University of Freiburg, Germany.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural