| pubmed-article:20177342 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20177342 | lifeskim:mentions | umls-concept:C0034493 | lld:lifeskim |
| pubmed-article:20177342 | lifeskim:mentions | umls-concept:C0332835 | lld:lifeskim |
| pubmed-article:20177342 | lifeskim:mentions | umls-concept:C0450127 | lld:lifeskim |
| pubmed-article:20177342 | lifeskim:mentions | umls-concept:C0741968 | lld:lifeskim |
| pubmed-article:20177342 | lifeskim:mentions | umls-concept:C0003850 | lld:lifeskim |
| pubmed-article:20177342 | lifeskim:mentions | umls-concept:C1514559 | lld:lifeskim |
| pubmed-article:20177342 | lifeskim:mentions | umls-concept:C0522536 | lld:lifeskim |
| pubmed-article:20177342 | lifeskim:mentions | umls-concept:C1553901 | lld:lifeskim |
| pubmed-article:20177342 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:20177342 | pubmed:dateCreated | 2010-2-23 | lld:pubmed |
| pubmed-article:20177342 | pubmed:abstractText | Cardiac transplant arteriosclerosis or cardiac allograft vasculopathy remains the leading cause of graft failure and patient death in heart transplant recipients. Endothelial cell injury is crucial in the development of human atherosclerosis and may play a role in allograft vasculopathy. Glutathione-S-transferase (GST) is known to protect endothelial cells from damage by oxidants and toxins. However, the contribution of human GST A4-4 (hGSTA4-4) to vascular cell injury and consequent transplant arteriosclerosis is unknown. | lld:pubmed |
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| pubmed-article:20177342 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20177342 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20177342 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:20177342 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20177342 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:20177342 | pubmed:issn | 1534-6080 | lld:pubmed |
| pubmed-article:20177342 | pubmed:author | pubmed-author:AwasthiYogesh... | lld:pubmed |
| pubmed-article:20177342 | pubmed:author | pubmed-author:BoorPaul JPJ | lld:pubmed |
| pubmed-article:20177342 | pubmed:author | pubmed-author:XuYaY | lld:pubmed |
| pubmed-article:20177342 | pubmed:author | pubmed-author:GongBinB | lld:pubmed |
| pubmed-article:20177342 | pubmed:author | pubmed-author:YangYongzhenY | lld:pubmed |
| pubmed-article:20177342 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20177342 | pubmed:day | 27 | lld:pubmed |
| pubmed-article:20177342 | pubmed:volume | 89 | lld:pubmed |
| pubmed-article:20177342 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20177342 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20177342 | pubmed:pagination | 409-16 | lld:pubmed |
| pubmed-article:20177342 | pubmed:dateRevised | 2011-9-26 | lld:pubmed |
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| pubmed-article:20177342 | pubmed:meshHeading | pubmed-meshheading:20177342... | lld:pubmed |
| pubmed-article:20177342 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20177342 | pubmed:articleTitle | Adenovirus-mediated overexpression of glutathione-s-transferase mitigates transplant arteriosclerosis in rabbit carotid allografts. | lld:pubmed |
| pubmed-article:20177342 | pubmed:affiliation | Department of Pathology, University of Texas Medical Branch, Galveston, TX, USA. | lld:pubmed |
| pubmed-article:20177342 | pubmed:publicationType | Journal Article | lld:pubmed |
