Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
4
pubmed:dateCreated
2010-2-23
pubmed:abstractText
Cardiac transplant arteriosclerosis or cardiac allograft vasculopathy remains the leading cause of graft failure and patient death in heart transplant recipients. Endothelial cell injury is crucial in the development of human atherosclerosis and may play a role in allograft vasculopathy. Glutathione-S-transferase (GST) is known to protect endothelial cells from damage by oxidants and toxins. However, the contribution of human GST A4-4 (hGSTA4-4) to vascular cell injury and consequent transplant arteriosclerosis is unknown.
pubmed:grant
pubmed:commentsCorrections
http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-10406932, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-11115475, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-11404178, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-11824376, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-12234961, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-12589154, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-12689622, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-12831811, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-12878982, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-12893000, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-12912909, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-12944567, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-12957609, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-14508350, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-14530191, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-15064094, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-15133765, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-15215168, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-15320847, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-15707403, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-15822171, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-15877800, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-16005854, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-16102427, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-16426308, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-17667816, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-18080870, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-18096479, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-18485437, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-19155974, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-2795279, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-7597707, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-8565162, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-8642787, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-8719476, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-8980630, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-9073611, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-9172260, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-9461507, http://linkedlifedata.com/resource/pubmed/commentcorrection/20177342-9772203
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1534-6080
pubmed:author
pubmed:issnType
Electronic
pubmed:day
27
pubmed:volume
89
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
409-16
pubmed:dateRevised
2011-9-26
pubmed:meshHeading
pubmed:year
2010
pubmed:articleTitle
Adenovirus-mediated overexpression of glutathione-s-transferase mitigates transplant arteriosclerosis in rabbit carotid allografts.
pubmed:affiliation
Department of Pathology, University of Texas Medical Branch, Galveston, TX, USA.
pubmed:publicationType
Journal Article
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