pubmed-article:20153825 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20153825 | lifeskim:mentions | umls-concept:C0016030 | lld:lifeskim |
pubmed-article:20153825 | lifeskim:mentions | umls-concept:C0521451 | lld:lifeskim |
pubmed-article:20153825 | lifeskim:mentions | umls-concept:C0005486 | lld:lifeskim |
pubmed-article:20153825 | lifeskim:mentions | umls-concept:C0439855 | lld:lifeskim |
pubmed-article:20153825 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:20153825 | pubmed:dateCreated | 2010-3-15 | lld:pubmed |
pubmed-article:20153825 | pubmed:abstractText | Complex I (CI) deficiency is the most frequent cause of OXPHOS disorders. Recent studies have shown increases in reactive oxygen species (ROS) production and mitochondrial network disturbances in patients' fibroblasts harbouring mutations in CI subunits. | lld:pubmed |
pubmed-article:20153825 | pubmed:language | eng | lld:pubmed |
pubmed-article:20153825 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20153825 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:20153825 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20153825 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:20153825 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20153825 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20153825 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20153825 | pubmed:month | May | lld:pubmed |
pubmed-article:20153825 | pubmed:issn | 0006-3002 | lld:pubmed |
pubmed-article:20153825 | pubmed:author | pubmed-author:MoránMM | lld:pubmed |
pubmed-article:20153825 | pubmed:author | pubmed-author:RiveraHH | lld:pubmed |
pubmed-article:20153825 | pubmed:author | pubmed-author:ArenasJJ | lld:pubmed |
pubmed-article:20153825 | pubmed:author | pubmed-author:MerineroBB | lld:pubmed |
pubmed-article:20153825 | pubmed:author | pubmed-author:MartínM AMA | lld:pubmed |
pubmed-article:20153825 | pubmed:author | pubmed-author:CuezvaJ MJM | lld:pubmed |
pubmed-article:20153825 | pubmed:author | pubmed-author:UgaldeCC | lld:pubmed |
pubmed-article:20153825 | pubmed:author | pubmed-author:BlázquezAA | lld:pubmed |
pubmed-article:20153825 | pubmed:author | pubmed-author:Sánchez-Aragó... | lld:pubmed |
pubmed-article:20153825 | pubmed:copyrightInfo | Copyright 2010 Elsevier B.V. All rights reserved. | lld:pubmed |
pubmed-article:20153825 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:20153825 | pubmed:volume | 1802 | lld:pubmed |
pubmed-article:20153825 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20153825 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20153825 | pubmed:pagination | 443-53 | lld:pubmed |
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pubmed-article:20153825 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20153825 | pubmed:articleTitle | Mitochondrial bioenergetics and dynamics interplay in complex I-deficient fibroblasts. | lld:pubmed |
pubmed-article:20153825 | pubmed:affiliation | Centro de Investigación, Hospital Universitario 12 de Octubre, Madrid, Spain. | lld:pubmed |
pubmed-article:20153825 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20153825 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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