20118201

Source:http://linkedlifedata.com/resource/pubmed/id/20118201

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0012655, umls-concept:C0020538, umls-concept:C0026809, umls-concept:C0205216, umls-concept:C0284725
pubmed:issue	1
pubmed:dateCreated	2010-6-14
pubmed:abstractText	By examining vasopressin V1a receptor (V1aR) knockout (KO) mice, we previously found that the V1aR is critically involved in the regulation of normal blood pressure. The present study was undertaken to elucidate the role of the V1aR in salt-induced hypertension.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0077427
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Adra1d protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Arginine Vasopressin, http://linkedlifedata.com/resource/pubmed/chemical/Hormone Antagonists, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Adrenergic, alpha-1, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Vasopressin, http://linkedlifedata.com/resource/pubmed/chemical/Saline Solution, Hypertonic, http://linkedlifedata.com/resource/pubmed/chemical/Sodium, http://linkedlifedata.com/resource/pubmed/chemical/Sodium Chloride, Dietary, http://linkedlifedata.com/resource/pubmed/chemical/vasopressin...
pubmed:status	MEDLINE
pubmed:month	Jul
pubmed:issn	1755-3245
pubmed:author	pubmed-author:DaichoTakuyaT, pubmed-author:HosodaChihiroC, pubmed-author:NasaYoshihisaY, pubmed-author:OikawaRyoR, pubmed-author:TakagiNorioN, pubmed-author:TakeoSatoshiS, pubmed-author:TanonakaKouichiK, pubmed-author:TanoueAkitoA, pubmed-author:TsujimotoGozohG
pubmed:issnType	Electronic
pubmed:day	1
pubmed:volume	87
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	187-94
pubmed:dateRevised	2010-11-18
pubmed:meshHeading	pubmed-meshheading:20118201-Animals, pubmed-meshheading:20118201-Arginine Vasopressin, pubmed-meshheading:20118201-Blood Pressure, pubmed-meshheading:20118201-Disease Models, Animal, pubmed-meshheading:20118201-Heart Rate, pubmed-meshheading:20118201-Hormone Antagonists, pubmed-meshheading:20118201-Hypertension, pubmed-meshheading:20118201-Injections, Intraventricular, pubmed-meshheading:20118201-Male, pubmed-meshheading:20118201-Mice, pubmed-meshheading:20118201-Mice, Inbred C57BL, pubmed-meshheading:20118201-Mice, Knockout, pubmed-meshheading:20118201-Nephrectomy, pubmed-meshheading:20118201-Receptors, Adrenergic, alpha-1, pubmed-meshheading:20118201-Receptors, Vasopressin, pubmed-meshheading:20118201-Saline Solution, Hypertonic, pubmed-meshheading:20118201-Sodium, pubmed-meshheading:20118201-Sodium Chloride, Dietary, pubmed-meshheading:20118201-Sympathetic Nervous System, pubmed-meshheading:20118201-Time Factors, pubmed-meshheading:20118201-Vasoconstriction
pubmed:year	2010
pubmed:articleTitle	Decreased susceptibility to salt-induced hypertension in subtotally nephrectomized mice lacking the vasopressin V1a receptor.
pubmed:affiliation	Department of Molecular and Cellular Pharmacology, Tokyo University of Pharmacy and Life Sciences, 1432-1, Horinouchi, Hachioji, Tokyo 192-0392, Japan.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't