| pubmed-article:20107181 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20107181 | lifeskim:mentions | umls-concept:C0017601 | lld:lifeskim |
| pubmed-article:20107181 | lifeskim:mentions | umls-concept:C0006784 | lld:lifeskim |
| pubmed-article:20107181 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:20107181 | lifeskim:mentions | umls-concept:C1517004 | lld:lifeskim |
| pubmed-article:20107181 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:20107181 | pubmed:dateCreated | 2010-5-20 | lld:pubmed |
| pubmed-article:20107181 | pubmed:abstractText | Glaucoma is a neurodegenerative disease in which elevated intraocular pressure (IOP) leads to progressive loss of retinal ganglion cells (RGCs) and blindness. Calcium dyshomeostasis has been suggested to play a role in the pathologic events that lead to RGC loss, though the details of these events are not well understood. Calcium-induced activation of calpain has been shown to contribute to neuronal death in a wide variety of neurodegenerative diseases. The authors hypothesize that similar events occur in glaucoma. | lld:pubmed |
| pubmed-article:20107181 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20107181 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20107181 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20107181 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20107181 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:20107181 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20107181 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20107181 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20107181 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:20107181 | pubmed:issn | 1552-5783 | lld:pubmed |
| pubmed-article:20107181 | pubmed:author | pubmed-author:HuangWeiW | lld:pubmed |
| pubmed-article:20107181 | pubmed:author | pubmed-author:GrosskreutzCy... | lld:pubmed |
| pubmed-article:20107181 | pubmed:author | pubmed-author:FiletaJohnJ | lld:pubmed |
| pubmed-article:20107181 | pubmed:author | pubmed-author:QuJuanJ | lld:pubmed |
| pubmed-article:20107181 | pubmed:author | pubmed-author:RaweIanI | lld:pubmed |
| pubmed-article:20107181 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20107181 | pubmed:volume | 51 | lld:pubmed |
| pubmed-article:20107181 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20107181 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20107181 | pubmed:pagination | 3049-54 | lld:pubmed |
| pubmed-article:20107181 | pubmed:dateRevised | 2011-3-3 | lld:pubmed |
| pubmed-article:20107181 | pubmed:meshHeading | pubmed-meshheading:20107181... | lld:pubmed |
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| pubmed-article:20107181 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20107181 | pubmed:articleTitle | Calpain activation in experimental glaucoma. | lld:pubmed |
| pubmed-article:20107181 | pubmed:affiliation | Howe Laboratory of Ophthalmology, Massachusetts Eye and Ear Infirmary, Boston, MA 02114, USA. | lld:pubmed |
| pubmed-article:20107181 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20107181 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:20107181 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
