19966310

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Subject	Predicate	Object	Context
pubmed-article:19966310	rdf:type	pubmed:Citation	lld:pubmed
pubmed-article:19966310	lifeskim:mentions	umls-concept:C0040300	lld:lifeskim
pubmed-article:19966310	lifeskim:mentions	umls-concept:C0001675	lld:lifeskim
pubmed-article:19966310	lifeskim:mentions	umls-concept:C0024660	lld:lifeskim
pubmed-article:19966310	lifeskim:mentions	umls-concept:C0034963	lld:lifeskim
pubmed-article:19966310	lifeskim:mentions	umls-concept:C0559956	lld:lifeskim
pubmed-article:19966310	lifeskim:mentions	umls-concept:C0449445	lld:lifeskim
pubmed-article:19966310	pubmed:issue	8	lld:pubmed
pubmed-article:19966310	pubmed:dateCreated	2010-2-25	lld:pubmed
pubmed-article:19966310	pubmed:abstractText	Urodeles and fetal mammals are capable of impressive epimorphic regeneration in a variety of tissues, whereas the typical default response to injury in adult mammals consists of inflammation and scar tissue formation. One component of epimorphic regeneration is the recruitment of resident progenitor and stem cells to a site of injury. Bioactive molecules resulting from degradation of extracellular matrix (ECM) have been shown to recruit a variety of progenitor and stem cells in vitro in adult mammals. The ability to recruit multipotential cells to the site of injury by in vivo administration of chemotactic ECM degradation products in a mammalian model of digit amputation was investigated in the present study. Adult, 6- to 8-week-old C57/BL6 mice were subjected to midsecond phalanx amputation of the third digit of the right hind foot and either treated with chemotactic ECM degradation products or left untreated. At 14 days after amputation, mice treated with ECM degradation products showed an accumulation of heterogeneous cells that expressed markers of multipotency, including Sox2, Sca1, and Rex1 (Zfp42). Cells isolated from the site of amputation were capable of differentiation along neuroectodermal and mesodermal lineages, whereas cells isolated from control mice were capable of differentiation along only mesodermal lineages. The present findings demonstrate the recruitment of endogenous stem cells to a site of injury, and/or their generation/proliferation therein, in response to ECM degradation products.	lld:pubmed
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pubmed-article:19966310	pubmed:language	eng	lld:pubmed
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pubmed-article:19966310	pubmed:status	MEDLINE	lld:pubmed
pubmed-article:19966310	pubmed:month	Feb	lld:pubmed
pubmed-article:19966310	pubmed:issn	1091-6490	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:TotteyStephen...	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:HirschiKaren...	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:ZhangLiL	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:WangGangG	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:GudasLorraine...	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:BadylakStephe...	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:JohnsonScott...	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:ReingJanetJ	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:BraunhutSusan...	lld:pubmed
pubmed-article:19966310	pubmed:author	pubmed-author:AgrawalVineet...	lld:pubmed
pubmed-article:19966310	pubmed:issnType	Electronic	lld:pubmed
pubmed-article:19966310	pubmed:day	23	lld:pubmed
pubmed-article:19966310	pubmed:volume	107	lld:pubmed
pubmed-article:19966310	pubmed:owner	NLM	lld:pubmed
pubmed-article:19966310	pubmed:authorsComplete	Y	lld:pubmed
pubmed-article:19966310	pubmed:pagination	3351-5	lld:pubmed
pubmed-article:19966310	pubmed:dateRevised	2011-8-4	lld:pubmed
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