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| pubmed-article:19940299 | lifeskim:mentions | umls-concept:C0033085 | lld:lifeskim |
| pubmed-article:19940299 | lifeskim:mentions | umls-concept:C0004561 | lld:lifeskim |
| pubmed-article:19940299 | lifeskim:mentions | umls-concept:C0393022 | lld:lifeskim |
| pubmed-article:19940299 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:19940299 | lifeskim:mentions | umls-concept:C0333668 | lld:lifeskim |
| pubmed-article:19940299 | pubmed:issue | 22 | lld:pubmed |
| pubmed-article:19940299 | pubmed:dateCreated | 2009-11-26 | lld:pubmed |
| pubmed-article:19940299 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19940299 | pubmed:abstractText | The immunopathogenesis of type 1 diabetes mellitus is associated with T-lymphocyte autoimmunity. However, there is growing evidence that B lymphocytes play a role in many T-lymphocyte-mediated diseases. It is possible to achieve selective depletion of B lymphocytes with rituximab, an anti-CD20 monoclonal antibody. This phase 2 study evaluated the role of B-lymphocyte depletion in patients with type 1 diabetes. | lld:pubmed |
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| pubmed-article:19940299 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19940299 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19940299 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:19940299 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19940299 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:19940299 | pubmed:issn | 1533-4406 | lld:pubmed |
| pubmed-article:19940299 | pubmed:author | pubmed-author:GolandRobinR | lld:pubmed |
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| pubmed-article:19940299 | pubmed:copyrightInfo | 2009 Massachusetts Medical Society | lld:pubmed |
| pubmed-article:19940299 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19940299 | pubmed:day | 26 | lld:pubmed |
| pubmed-article:19940299 | pubmed:volume | 361 | lld:pubmed |
| pubmed-article:19940299 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19940299 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19940299 | pubmed:pagination | 2143-52 | lld:pubmed |
| pubmed-article:19940299 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:19940299 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19940299 | pubmed:articleTitle | Rituximab, B-lymphocyte depletion, and preservation of beta-cell function. | lld:pubmed |
| pubmed-article:19940299 | pubmed:affiliation | Indiana University School of Medicine, Indianapolis, USA. | lld:pubmed |
| pubmed-article:19940299 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19940299 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
| pubmed-article:19940299 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:19940299 | pubmed:publicationType | Multicenter Study | lld:pubmed |
| pubmed-article:19940299 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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