| pubmed-article:1981057 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1981057 | lifeskim:mentions | umls-concept:C0013336 | lld:lifeskim |
| pubmed-article:1981057 | lifeskim:mentions | umls-concept:C0812327 | lld:lifeskim |
| pubmed-article:1981057 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:1981057 | lifeskim:mentions | umls-concept:C0084828 | lld:lifeskim |
| pubmed-article:1981057 | lifeskim:mentions | umls-concept:C0591833 | lld:lifeskim |
| pubmed-article:1981057 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:1981057 | pubmed:dateCreated | 1991-3-25 | lld:pubmed |
| pubmed-article:1981057 | pubmed:abstractText | Two nonallelic mouse mutations with severe dwarf phenotypes are characterized by a lack of growth hormone, prolactin, and thyroid stimulating hormone. The cells that normally synthesize these pituitary hormones express a common transcription factor called GHF-1 or Pit-1. Using an intersubspecific backcross, we have demonstrated tight linkage of the Pit-1 and Snell dwarf (dw) genes on mouse chromosome 16. No recombination was observed between Pit-1 and dw in 110 individuals examined. Southern blot analysis of genomic DNA reveals that the Pit-1 gene is rearranged in C3H/HeJ-dwJ/dw mice but not in coisogenic +/+ animals, providing molecular evidence that a lesion in the Pit-1 gene results in the Snell dwarf phenotype. Demonstration of low levels of Pit-1 expression in Ames dwarf (df) mice implies that both Pit-1 and df expression may be required for pituitary differentiation. | lld:pubmed |
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| pubmed-article:1981057 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1981057 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1981057 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1981057 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1981057 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1981057 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:1981057 | pubmed:issn | 0888-7543 | lld:pubmed |
| pubmed-article:1981057 | pubmed:author | pubmed-author:ReevesR HRH | lld:pubmed |
| pubmed-article:1981057 | pubmed:author | pubmed-author:CamperS ASA | lld:pubmed |
| pubmed-article:1981057 | pubmed:author | pubmed-author:KatzR WRW | lld:pubmed |
| pubmed-article:1981057 | pubmed:author | pubmed-author:SaundersT LTL | lld:pubmed |
| pubmed-article:1981057 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1981057 | pubmed:volume | 8 | lld:pubmed |
| pubmed-article:1981057 | pubmed:geneSymbol | df | lld:pubmed |
| pubmed-article:1981057 | pubmed:geneSymbol | dw | lld:pubmed |
| pubmed-article:1981057 | pubmed:geneSymbol | dw<up>J</up> | lld:pubmed |
| pubmed-article:1981057 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1981057 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1981057 | pubmed:pagination | 586-90 | lld:pubmed |
| pubmed-article:1981057 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:1981057 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:1981057 | pubmed:articleTitle | The Pit-1 transcription factor gene is a candidate for the murine Snell dwarf mutation. | lld:pubmed |
| pubmed-article:1981057 | pubmed:affiliation | Department of Human Genetics, University of Michigan Medical School, Ann Arbor 48109-0618. | lld:pubmed |
| pubmed-article:1981057 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1981057 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:1981057 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:1981057 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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