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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2009-9-22
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pubmed:abstractText |
Hydrogen sulfide is produced endogenously in response to myocardial ischemia and thought to be cardioprotective. The mechanism underlying this protection has yet to be fully elucidated, but it may be related to sulfide's ability to limit inflammation. This study investigates the cardioprotection provided by exogenous hydrogen sulfide and its potential anti-inflammatory mechanism of action.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
1097-685X
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
138
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
977-84
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pubmed:dateRevised |
2010-12-28
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pubmed:meshHeading |
pubmed-meshheading:19660398-Animals,
pubmed-meshheading:19660398-Anti-Inflammatory Agents,
pubmed-meshheading:19660398-Coronary Vessels,
pubmed-meshheading:19660398-Cytokines,
pubmed-meshheading:19660398-Free Radicals,
pubmed-meshheading:19660398-Hydrogen Sulfide,
pubmed-meshheading:19660398-Immunohistochemistry,
pubmed-meshheading:19660398-Microvessels,
pubmed-meshheading:19660398-Myocardial Contraction,
pubmed-meshheading:19660398-Myocardial Infarction,
pubmed-meshheading:19660398-Myocardial Reperfusion Injury,
pubmed-meshheading:19660398-Myocardium,
pubmed-meshheading:19660398-Peroxidase,
pubmed-meshheading:19660398-Swine
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pubmed:year |
2009
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pubmed:articleTitle |
Hydrogen sulfide therapy attenuates the inflammatory response in a porcine model of myocardial ischemia/reperfusion injury.
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pubmed:affiliation |
Division of Cardiothoracic Surgery, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Mass., USA.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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