| pubmed-article:19590190 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19590190 | lifeskim:mentions | umls-concept:C0858252 | lld:lifeskim |
| pubmed-article:19590190 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:19590190 | lifeskim:mentions | umls-concept:C0032824 | lld:lifeskim |
| pubmed-article:19590190 | lifeskim:mentions | umls-concept:C0205617 | lld:lifeskim |
| pubmed-article:19590190 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:19590190 | lifeskim:mentions | umls-concept:C0376452 | lld:lifeskim |
| pubmed-article:19590190 | lifeskim:mentions | umls-concept:C0086860 | lld:lifeskim |
| pubmed-article:19590190 | pubmed:issue | 1-2 | lld:pubmed |
| pubmed-article:19590190 | pubmed:dateCreated | 2009-7-10 | lld:pubmed |
| pubmed-article:19590190 | pubmed:abstractText | DNA methylation is an important mechanism for gene silencing and has already been described for several genes in breast cancer. A previous immunohistochemistry study demonstrated a decrease of K(v)1.3 potassium channel expression in breast adenocarcinoma compared to normal breast tissue. | lld:pubmed |
| pubmed-article:19590190 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19590190 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19590190 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19590190 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19590190 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19590190 | pubmed:issn | 1421-9778 | lld:pubmed |
| pubmed-article:19590190 | pubmed:author | pubmed-author:SevestreHenri... | lld:pubmed |
| pubmed-article:19590190 | pubmed:author | pubmed-author:MervielPhilip... | lld:pubmed |
| pubmed-article:19590190 | pubmed:author | pubmed-author:Ouadid-Ahidou... | lld:pubmed |
| pubmed-article:19590190 | pubmed:author | pubmed-author:BrevetMarieM | lld:pubmed |
| pubmed-article:19590190 | pubmed:author | pubmed-author:HarenNathalie... | lld:pubmed |
| pubmed-article:19590190 | pubmed:copyrightInfo | 2009 S. Karger AG, Basel. | lld:pubmed |
| pubmed-article:19590190 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19590190 | pubmed:volume | 24 | lld:pubmed |
| pubmed-article:19590190 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19590190 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19590190 | pubmed:pagination | 25-32 | lld:pubmed |
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| pubmed-article:19590190 | pubmed:meshHeading | pubmed-meshheading:19590190... | lld:pubmed |
| pubmed-article:19590190 | pubmed:meshHeading | pubmed-meshheading:19590190... | lld:pubmed |
| pubmed-article:19590190 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19590190 | pubmed:articleTitle | DNA methylation of K(v)1.3 potassium channel gene promoter is associated with poorly differentiated breast adenocarcinoma. | lld:pubmed |
| pubmed-article:19590190 | pubmed:affiliation | Laboratoire de Physiologie Cellulaire et Moléculaire, Faculté des Sciences, Amiens, France. | lld:pubmed |
| pubmed-article:19590190 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19590190 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:3738 | entrezgene:pubmed | pubmed-article:19590190 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:19590190 | lld:entrezgene |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:19590190 | lld:pubmed |
