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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1-2
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pubmed:dateCreated |
2009-7-10
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pubmed:abstractText |
DNA methylation is an important mechanism for gene silencing and has already been described for several genes in breast cancer. A previous immunohistochemistry study demonstrated a decrease of K(v)1.3 potassium channel expression in breast adenocarcinoma compared to normal breast tissue.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:issn |
1421-9778
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pubmed:author |
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pubmed:copyrightInfo |
2009 S. Karger AG, Basel.
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pubmed:issnType |
Electronic
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pubmed:volume |
24
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|
pubmed:owner |
NLM
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|
pubmed:authorsComplete |
Y
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pubmed:pagination |
25-32
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pubmed:meshHeading |
pubmed-meshheading:19590190-Adenocarcinoma,
pubmed-meshheading:19590190-Adult,
pubmed-meshheading:19590190-Aged,
pubmed-meshheading:19590190-Breast Neoplasms,
pubmed-meshheading:19590190-DNA Methylation,
pubmed-meshheading:19590190-Female,
pubmed-meshheading:19590190-Humans,
pubmed-meshheading:19590190-Kv1.3 Potassium Channel,
pubmed-meshheading:19590190-Middle Aged,
pubmed-meshheading:19590190-Neoplasm Staging,
pubmed-meshheading:19590190-Polymerase Chain Reaction,
pubmed-meshheading:19590190-Promoter Regions, Genetic,
pubmed-meshheading:19590190-RNA, Messenger,
pubmed-meshheading:19590190-Tumor Cells, Cultured
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pubmed:year |
2009
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pubmed:articleTitle |
DNA methylation of K(v)1.3 potassium channel gene promoter is associated with poorly differentiated breast adenocarcinoma.
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pubmed:affiliation |
Laboratoire de Physiologie Cellulaire et Moléculaire, Faculté des Sciences, Amiens, France.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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